Respiratory tract infections increase susceptibility to neurogenic inflammation in the rat trachea.
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Airway vasculature after mycoplasma infection: chronic leakiness and selective hypersensitivity to substance P.Hypertonicity, but not hypothermia, elicits substance P release from rat C-fiber neurons in primary cultureNeutral endopeptidase and kininase II mediate glucocorticoid inhibition of neurogenic inflammation in the rat trachea.On lung nerves and neurogenic injury.Neurogenic inflammation and asthma.Cigarette smoke induces bronchoconstrictor hyperresponsiveness to substance P and inactivates airway neutral endopeptidase in the guinea pig. Possible role of free radicalsMycoplasma pulmonis infections cause long-lasting potentiation of neurogenic inflammation in the respiratory tract of the ratEffects of aerosolised substance P on lung resistance in guinea-pigs: a comparison between inhibition of neutral endopeptidase and angiotensin-converting enzymeViral infection induces dependence of neuronal M2 muscarinic receptors on cyclooxygenase in guinea pig lung.Sensory nerves, neuropeptides, and asthma.Glucocorticoids inhibit neurogenic plasma extravasation and prevent virus-potentiated extravasation in the rat trachea.New perspectives on basic mechanisms in lung disease. 4. Why are the airways so vascular?Tachykinin peptides, receptors, and peptidases in airway disease.Repeated challenge with dinitrobenzene sulphonic acid in dinitrofluorobenzene-sensitized mice results in vascular hyperpermeability in the trachea: a role for tachykinins.Increase in vascular permeability produced in rat airways by PAF: potentiation by adrenalectomyBronchial responses to substance P after antigen challenge in the guinea-pig: in vivo and in vitro studies.Histamine-induced changes in rat tracheal goblet cell mucin store and mucosal edema.Spontaneous remission of edema and regranulation of goblet cells in rat tracheae after capsaicin-induced acute inflammation.Changes in airway reactivity to exogenous and endogenous acetylcholine and substance P after anaphylactic bronchoconstriction in anaesthetized guinea-pigs.Virus-induced airway hyperresponsiveness and asthma.Characterization of upper respiratory disease in rats following neonatal inoculation with a rat-adapted influenza virus.Brown Norway rats are high responders to bronchiolitis, pneumonia, and bronchiolar mastocytosis induced by parainfluenza virus.Substance P-immunoreactive sensory axons in the rat respiratory tract: a quantitative study of their distribution and role in neurogenic inflammation.Three-dimensional visualization of blood and lymphatic vasculature in tissue whole mounts using confocal microscopy.Hyperosmolar saline induces airway resistance changes and neuropeptide release: a comparison with the effect of capsaicin, potassium and histamine.Occurrence of mast cells in relation to the distribution of nerve fibers in the rat larynx.
P2860
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P2860
Respiratory tract infections increase susceptibility to neurogenic inflammation in the rat trachea.
description
1988 nî lūn-bûn
@nan
1988年の論文
@ja
1988年学术文章
@wuu
1988年学术文章
@zh
1988年学术文章
@zh-cn
1988年学术文章
@zh-hans
1988年学术文章
@zh-my
1988年学术文章
@zh-sg
1988年學術文章
@yue
1988年學術文章
@zh-hant
name
Respiratory tract infections i ...... flammation in the rat trachea.
@en
Respiratory tract infections i ...... flammation in the rat trachea.
@nl
type
label
Respiratory tract infections i ...... flammation in the rat trachea.
@en
Respiratory tract infections i ...... flammation in the rat trachea.
@nl
prefLabel
Respiratory tract infections i ...... flammation in the rat trachea.
@en
Respiratory tract infections i ...... flammation in the rat trachea.
@nl
P356
P1476
Respiratory tract infections i ...... flammation in the rat trachea.
@en
P2093
McDonald DM
P304
P356
10.1164/AJRCCM/137.6.1432
P577
1988-06-01T00:00:00Z