Targeted disruption of the meprin metalloproteinase beta gene protects against renal ischemia-reperfusion injury in mice.
about
Meprins, membrane-bound and secreted astacin metalloproteinasesADAM10 is the major sheddase responsible for the release of membrane-associated meprin A.Urinary signatures of Renal Cell Carcinoma investigated by peptidomic approaches.Balance of meprin A and B in mice affects the progression of experimental inflammatory bowel disease.Isoform-specific interactions between meprin metalloproteases and the catalytic subunit of protein kinase A: significance in acute and chronic kidney injury.Endogenous IL-10 attenuates cisplatin nephrotoxicity: role of dendritic cellsVillin and actin in the mouse kidney brush-border membrane bind to and are degraded by meprins, an interaction that contributes to injury in ischemia-reperfusionHyperoxia-Induced Protein Alterations in Renal Rat Tissue: A Quantitative Proteomic Approach to Identify Hyperoxia-Induced Effects in Cellular Signaling Pathways.The metalloproteases meprin α and meprin β: unique enzymes in inflammation, neurodegeneration, cancer and fibrosis.Meprin A metalloproteinase and its role in acute kidney injury.The anti-inflammatory peptide Ac-SDKP is released from thymosin-β4 by renal meprin-α and prolyl oligopeptidase.Meprin A metalloproteases enhance renal damage and bladder inflammation after LPS challenge.Hypoxia Associated Proteolytic Processing of OS-9 by the Metalloproteinase Meprin β.Meprin A impairs epithelial barrier function, enhances monocyte migration, and cleaves the tight junction protein occludin.Protective effects of ezrin on cold storage preservation injury in the pig kidney proximal tubular epithelial cell line (LLC-PK1)Role of GLP-1 and DPP-4 in diabetic nephropathy and cardiovascular disease.Renal Effects of DPP-4 Inhibitors: A Focus on MicroalbuminuriaThe Metalloprotease Meprin β Is an Alternative β-Secretase of APP.Meprin Metalloprotease Deficiency Associated with Higher Mortality Rates and More Severe Diabetic Kidney Injury in Mice with STZ-Induced Type 1 Diabetes.Basement membrane protein nidogen-1 is a target of meprin β in cisplatin nephrotoxicity.Local overexpression of interleukin-1 family, member 6 relates to the development of tubulointerstitial lesions.
P2860
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P2860
Targeted disruption of the meprin metalloproteinase beta gene protects against renal ischemia-reperfusion injury in mice.
description
2008 nî lūn-bûn
@nan
2008年の論文
@ja
2008年学术文章
@wuu
2008年学术文章
@zh
2008年学术文章
@zh-cn
2008年学术文章
@zh-hans
2008年学术文章
@zh-my
2008年学术文章
@zh-sg
2008年學術文章
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2008年學術文章
@zh-hant
name
Targeted disruption of the mep ...... ia-reperfusion injury in mice.
@en
Targeted disruption of the mep ...... ia-reperfusion injury in mice.
@nl
type
label
Targeted disruption of the mep ...... ia-reperfusion injury in mice.
@en
Targeted disruption of the mep ...... ia-reperfusion injury in mice.
@nl
prefLabel
Targeted disruption of the mep ...... ia-reperfusion injury in mice.
@en
Targeted disruption of the mep ...... ia-reperfusion injury in mice.
@nl
P2093
P2860
P1476
Targeted disruption of the mep ...... mia-reperfusion injury in mice
@en
P2093
Binzhi Zhang
Ganesan Ramesh
John Bylander
Judith S Bond
P2860
P304
P356
10.1152/AJPRENAL.00214.2007
P50
P577
2008-01-02T00:00:00Z