Recurrent abortions in Asian Indians: no role of factor V Leiden Hong Kong/Cambridge mutation and MTHFR polymorphism. - Wikidata - awgldk - TriplyDB

Recurrent abortions in Asian Indians: no role of factor V Leiden Hong Kong/Cambridge mutation and MTHFR polymorphism.

Recurrent abortions in Asian Indians: no role of factor V Leiden Hong Kong/Cambridge mutation and MTHFR polymorphism.

http://www.wikidata.org/entity/Q46832268

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Prevalence of thromogenic gene mutations in women with recurrent miscarriage: A retrospective study of 1,507 patients.Ethnic differences in the association of thrombophilic polymorphisms with obstetric complications in Slovak and Roma (Gypsy) populations Evaluation of chromosomal abnormalities and common trombophilic mutations in cases with recurrent miscarriage.Association between maternal, fetal and paternal MTHFR gene C677T and A1298C polymorphisms and risk of recurrent pregnancy loss: a comprehensive evaluation.Inherited thrombophilia profile in patients with recurrent miscarriages: Experience from a tertiary care center in north India.Can Factor V Leiden and prothrombin G20210A testing in women with recurrent pregnancy loss result in improved pregnancy outcomes?: Results from a targeted evidence-based review.Association of Plasminogen Activator Inhibitor-1 Gene Polymorphisms and Methylene Tetrahydrofolate Reductase Polymorphisms with Spontaneous Miscarriages.Association of Methylenetetrahydrofolate Reductase C677T and A1298C Gene Polymorphisms With Recurrent Pregnancy Loss in Syrian Women.Candidate gene study of genetic thrombophilic polymorphisms in pre-eclampsia and recurrent pregnancy loss in Sinhalese women.MTHFR C677T polymorphism and recurrent early pregnancy loss risk in north Indian population.Analysis of plasminogen activator inhibitor-1, integrin beta3, beta fibrinogen, and methylenetetrahydrofolate reductase polymorphisms in Iranian women with recurrent pregnancy loss.Lack of association between thrombophilic gene variants and recurrent pregnancy loss Plasminogen activator inhibitor-1, factor V, factor II and methylenetetrahydrofolate reductase polymorphisms in women with recurrent miscarriage

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P2860

Recurrent abortions in Asian Indians: no role of factor V Leiden Hong Kong/Cambridge mutation and MTHFR polymorphism.

http://www.wikidata.org/entity/Q46832268

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2007 nî lūn-bûn

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2007年の論文

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2007年学术文章

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2007年学术文章

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2007年学术文章

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2007年学术文章

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2007年学术文章

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2007年學術文章

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2007年學術文章

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name

Recurrent abortions in Asian I ...... tation and MTHFR polymorphism.

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Recurrent abortions in Asian I ...... tation and MTHFR polymorphism.

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Recurrent abortions in Asian I ...... tation and MTHFR polymorphism.

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Recurrent abortions in Asian I ...... tation and MTHFR polymorphism.

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Recurrent abortions in Asian I ...... tation and MTHFR polymorphism.

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Recurrent abortions in Asian I ...... tation and MTHFR polymorphism.

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Clinical and Applied Thrombosis-Hemostasis

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Recurrent abortions in Asian I ...... utation and MTHFR polymorphism

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Anuradha Mittal

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Arvind Meena

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Priya Choudhry

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Ravi Ranjan

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Renu Saxena

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V P Choudhry

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P2860

A candidate genetic risk factor for vascular disease: a common mutation in methylenetetrahydrofolate reductase

Prevalence of antiphospholipid antibodies, factor V G1691A (Leiden) and prothrombin G20210A mutations in early and late recurrent pregnancy loss.

Search for factor V Arg306 Cambridge and Hong Kong mutations in mixed Hungarian population samples.

MTHFR C677T Polymorphism and factor V Leiden mutation are not associated with recurrent spontaneous abortion of unexplained etiology in Japanese women.

Association of anticardiolipin antibody and C677T in methylenetetrahydrofolate reductase mutation in women with recurrent spontaneous abortions: a new path to thrombophilia?

Recurrent pregnancy loss and its relation to FV Leiden, FII G20210A and polymorphisms of plasminogen activator and plasminogen activator inhibitor.

Factor V Leiden mutation as a risk factor for recurrent pregnancy loss.

High prevalence of a mutation in the factor V gene within the U.K. population: relationship to activated protein C resistance and familial thrombosis.

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