A20 controls intestinal homeostasis through cell-specific activities. - Wikidata - awgldk - TriplyDB

A20 controls intestinal homeostasis through cell-specific activities.

A20 controls intestinal homeostasis through cell-specific activities.

http://www.wikidata.org/entity/Q46832278

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Genetically engineered mouse models for studying inflammatory bowel disease Drug Discovery via Human-Derived Stem Cell Organoids A20 Inhibits β-Cell Apoptosis by Multiple Mechanisms and Predicts Residual β-Cell Function in Type 1 Diabetes.Inflammation, metaflammation and immunometabolic disorders.How informative is the mouse for human gut microbiota research?Physiological relevance of food grade microcapsules: Impact of milk protein based microcapsules on inflammation in mouse models for inflammatory bowel diseases.Regulatory immune cells in regulation of intestinal inflammatory response to microbiota.Normalization of Host Intestinal Mucus Layers Requires Long-Term Microbial Colonization Epithelial-intrinsic IKKα expression regulates group 3 innate lymphoid cell responses and antibacterial immunity.A20 prevents chronic liver inflammation and cancer by protecting hepatocytes from death.Enhanced Mucosal Defense and Reduced Tumor Burden in Mice with the Compromised Negative Regulator IRAK-M.Altered expression of A20 gene in peripheral blood mononuclear cells is associated with the progression of chronic hepatitis B virus infection.CYLD, A20 and OTULIN deubiquitinases in NF-κB signaling and cell death: so similar, yet so different.Limiting inflammation-the negative regulation of NF-κB and the NLRP3 inflammasome.A20, an essential component of the ubiquitin-editing protein complex, is a negative regulator of inflammation in human myometrium and foetal membranes.Epithelial EZH2 serves as an epigenetic determinant in experimental colitis by inhibiting TNFα-mediated inflammation and apoptosis.NOD2 and bacterial recognition as therapeutic targets for Crohn's disease.Immune Regulation by Ubiquitin Tagging as Checkpoint Code.Inflammatory cell death in intestinal pathologies.A20 regulates canonical wnt-signaling through an interaction with RIPK4.Tumor necrosis factor α-induced protein 3 (A20) is dysregulated in pediatric Crohn disease.Regulation of Nuclear Factor Kappa-Light-Chain-Enhancer of Activated B Cells (NF-κβ) in Inflammatory Bowel Diseases

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A20 controls intestinal homeostasis through cell-specific activities.

http://www.wikidata.org/entity/Q46832278

description

2014 nî lūn-bûn

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2014年の論文

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2014年学术文章

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2014年学术文章

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2014年学术文章

@zh-cn

2014年学术文章

@zh-hans

2014年学术文章

@zh-my

2014年学术文章

@zh-sg

2014年學術文章

@yue

2014年學術文章

@zh-hant

name

A20 controls intestinal homeostasis through cell-specific activities.

@en

A20 controls intestinal homeostasis through cell-specific activities.

@nl

type

label

A20 controls intestinal homeostasis through cell-specific activities.

@en

A20 controls intestinal homeostasis through cell-specific activities.

@nl

prefLabel

A20 controls intestinal homeostasis through cell-specific activities.

@en

A20 controls intestinal homeostasis through cell-specific activities.

@nl

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Nature Communications

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A20 controls intestinal homeostasis through cell-specific activities.

@en

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Conor Mc Guire

http://www.w3.org/2001/XMLSchema#string

Geert van Loo

http://www.w3.org/2001/XMLSchema#string

Gert De Hertogh

http://www.w3.org/2001/XMLSchema#string

Hans Clevers

http://www.w3.org/2001/XMLSchema#string

Johan H van Es

http://www.w3.org/2001/XMLSchema#string

Karolina Slowicka

http://www.w3.org/2001/XMLSchema#string

Lars Vereecke

http://www.w3.org/2001/XMLSchema#string

Maaike van den Born

http://www.w3.org/2001/XMLSchema#string

Mozes Sze

http://www.w3.org/2001/XMLSchema#string

Thomas Billiet

http://www.w3.org/2001/XMLSchema#string

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Host-microbe interactions have shaped the genetic architecture of inflammatory bowel disease

XBP1 links ER stress to intestinal inflammation and confers genetic risk for human inflammatory bowel disease

Intestinal tumorigenesis initiated by dedifferentiation and acquisition of stem-cell-like properties

Genome-wide association study of 14,000 cases of seven common diseases and 3,000 shared controls

Virus-plus-susceptibility gene interaction determines Crohn's disease gene Atg16L1 phenotypes in intestine

QIIME allows analysis of high-throughput community sequencing data

Paneth cells directly sense gut commensals and maintain homeostasis at the intestinal host-microbial interface

PLINK: a tool set for whole-genome association and population-based linkage analyses

Communicable ulcerative colitis induced by T-bet deficiency in the innate immune system

Cancer-related inflammation

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5103

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scholarly article

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10.1038/NCOMMS6103

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5

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Séverine Vermeire

Sara Vieira-Silva

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2014-09-30T00:00:00Z

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266324434

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25267258

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