Calcium spark properties in ventricular myocytes are altered in aged mice.
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Recapitulating maladaptive, multiscale remodeling of failing myocardium on a chip.Early development of intracellular calcium cycling defects in intact hearts of spontaneously hypertensive ratsModulation of sarcoplasmic reticulum Ca(2+) cycling in systolic and diastolic heart failure associated with aging.Dantrolene suppresses spontaneous Ca2+ release without altering excitation-contraction coupling in cardiomyocytes of aged mice.Functional role of Calstabin2 in age-related cardiac alterations.Age-dependent dystrophin loss and genetic reconstitution establish a molecular link between dystrophin and heart performance during aging.Effect of aging on power output properties in rat skinned cardiac myocytes.JTV519 (K201) reduces sarcoplasmic reticulum Ca²⁺ leak and improves diastolic function in vitro in murine and human non-failing myocardiummTOR pathway and Ca²⁺ stores mobilization in aged smooth muscle cellsAltered Ca2+ sparks in aging skeletal and cardiac muscle.Interactions of Short-Term and Chronic Treadmill Training With Aging of the Left Ventricle of the HeartCardiac electrical defects in progeroid mice and Hutchinson-Gilford progeria syndrome patients with nuclear lamina alterations.The SR-mitochondria interaction: a new player in cardiac pathophysiology.Pathways for salvage and protection of the heart under stress: novel routes for cardiac rejuvenation.Cardiomyocyte Ca2+ homeostasis as a therapeutic target in heart failure with reduced and preserved ejection fraction.Propagation of Intracellular Ca2+ Signals in Aged Exocrine Cells.Age-related regulation of excitation-contraction coupling in rat heart.The effects of aging on the regulation of t-tubular ICa by caveolin in mouse ventricular myocytes.Left Atrial Volume and Pulmonary Artery Diameter Are Noninvasive Measures of Age-Related Diastolic Dysfunction in Mice.Age-associated changes in excitation-contraction coupling are more prominent in ventricular myocytes from male rats than in myocytes from female rats.NOX Inhibition Improves β-Adrenergic Stimulated Contractility and Intracellular Calcium Handling in the Aged Rat Heart
P2860
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P2860
Calcium spark properties in ventricular myocytes are altered in aged mice.
description
2005 nî lūn-bûn
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2005年の論文
@ja
2005年学术文章
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2005年学术文章
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2005年学术文章
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2005年学术文章
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2005年学术文章
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2005年學術文章
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2005年學術文章
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name
Calcium spark properties in ventricular myocytes are altered in aged mice.
@en
Calcium spark properties in ventricular myocytes are altered in aged mice.
@nl
type
label
Calcium spark properties in ventricular myocytes are altered in aged mice.
@en
Calcium spark properties in ventricular myocytes are altered in aged mice.
@nl
prefLabel
Calcium spark properties in ventricular myocytes are altered in aged mice.
@en
Calcium spark properties in ventricular myocytes are altered in aged mice.
@nl
P2860
P1476
Calcium spark properties in ventricular myocytes are altered in aged mice.
@en
P2093
Gregory R Ferrier
Scott A Grandy
P2860
P304
P356
10.1152/AJPHEART.00686.2005
P577
2005-12-03T00:00:00Z