Catalase T and Cu,Zn-superoxide dismutase in the acetic acid-induced programmed cell death in Saccharomyces cerevisiae.
about
The expanding role of yeast in cancer research and diagnosis: insights into the function of the oncosuppressors p53 and BRCA1/2The role of mitochondria in yeast programmed cell deathCatalase overexpression reduces lactic acid-induced oxidative stress in Saccharomyces cerevisiaeUntangling the Roles of Anti-Apoptosis in Regulating Programmed Cell Death using Humanized Yeast Cells.Modulation of mitochondrial outer membrane permeabilization and apoptosis by ceramide metabolism.Improvement of acetic acid tolerance of Saccharomyces cerevisiae using a zinc-finger-based artificial transcription factor and identification of novel genes involved in acetic acid tolerance.Overexpression of acetyl-CoA synthetase in Saccharomyces cerevisiae increases acetic acid tolerance.Mitochondrial proteomics of the acetic acid - induced programmed cell death response in a highly tolerant Zygosaccharomyces bailii - derived hybrid strain.Achievements and perspectives in yeast acetic acid-induced programmed cell death pathways.Silencing of BRCA2 decreases anoikis and its heterologous expression sensitizes yeast cells to acetic acid-induced programmed cell death.Yeast colony survival depends on metabolic adaptation and cell differentiation rather than on stress defenseMolecular mechanisms of Saccharomyces cerevisiae stress adaptation and programmed cell death in response to acetic acidYcf1p attenuates basal level oxidative stress response in Saccharomyces cerevisiae.Knock-out of metacaspase and/or cytochrome c results in the activation of a ROS-independent acetic acid-induced programmed cell death pathway in yeast.Lack of HXK2 induces localization of active Ras in mitochondria and triggers apoptosis in the yeast Saccharomyces cerevisiae.Yeast acetic acid-induced programmed cell death can occur without cytochrome c release which requires metacaspase YCA1.Different response to acetic acid stress in Saccharomyces cerevisiae wild-type and l-ascorbic acid-producing strains.Loss of peroxisome function triggers necrosis.Improved growth and ethanol fermentation of Saccharomyces cerevisiae in the presence of acetic acid by overexpression of SET5 and PPR1.Cytochrome c is released from coupled mitochondria of yeast en route to acetic acid-induced programmed cell death and can work as an electron donor and a ROS scavenger.Absence of Rtt109p, a fungal-specific histone acetyltransferase, results in improved acetic acid tolerance of Saccharomyces cerevisiae.Yeast as a tool to study signaling pathways in mitochondrial stress response and cytoprotection
P2860
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P2860
Catalase T and Cu,Zn-superoxide dismutase in the acetic acid-induced programmed cell death in Saccharomyces cerevisiae.
description
2007 nî lūn-bûn
@nan
2007年の論文
@ja
2007年学术文章
@wuu
2007年学术文章
@zh-cn
2007年学术文章
@zh-hans
2007年学术文章
@zh-my
2007年学术文章
@zh-sg
2007年學術文章
@yue
2007年學術文章
@zh
2007年學術文章
@zh-hant
name
Catalase T and Cu,Zn-superoxid ...... h in Saccharomyces cerevisiae.
@en
Catalase T and Cu,Zn-superoxid ...... h in Saccharomyces cerevisiae.
@nl
type
label
Catalase T and Cu,Zn-superoxid ...... h in Saccharomyces cerevisiae.
@en
Catalase T and Cu,Zn-superoxid ...... h in Saccharomyces cerevisiae.
@nl
prefLabel
Catalase T and Cu,Zn-superoxid ...... h in Saccharomyces cerevisiae.
@en
Catalase T and Cu,Zn-superoxid ...... h in Saccharomyces cerevisiae.
@nl
P2093
P2860
P1433
P1476
Catalase T and Cu,Zn-superoxid ...... h in Saccharomyces cerevisiae.
@en
P2093
Ersilia Marra
Lucia Antonacci
Nicoletta Guaragnella
Salvatore Passarella
P2860
P304
P356
10.1016/J.FEBSLET.2007.12.007
P407
P577
2007-12-17T00:00:00Z