Hyperoxia-induced reactive oxygen species formation in pulmonary capillary endothelial cells in situ. - Wikidata - awgldk - TriplyDB

Hyperoxia-induced reactive oxygen species formation in pulmonary capillary endothelial cells in situ.

Hyperoxia-induced reactive oxygen species formation in pulmonary capillary endothelial cells in situ.

http://www.wikidata.org/entity/Q46853361

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Effects of Hyperoxia on Oxygen-Related Inflammation with a Focus on Obesity Swimbladder function and the spawning migration of the European eel Anguilla anguilla.Reactive oxygen species-dependent RhoA activation mediates collagen synthesis in hyperoxic lung fibrosis Oxidative lipidomics of hyperoxic acute lung injury: mass spectrometric characterization of cardiolipin and phosphatidylserine peroxidation Hyperoxia-induced lung injury is dose dependent in Wistar rats Normobaric hyperoxia therapy for traumatic brain injury and stroke: a review.Oxygen delivery, consumption, and conversion to reactive oxygen species in experimental models of diabetic retinopathy Role of Nox4 and Nox2 in hyperoxia-induced reactive oxygen species generation and migration of human lung endothelial cells.The Risk of Oxygen during Cardiac Surgery (ROCS) trial: study protocol for a randomized clinical trial eNOS-beta-actin interaction contributes to increased peroxynitrite formation during hyperoxia in pulmonary artery endothelial cells and mouse lungs.Interactions between calcium and reactive oxygen species in pulmonary arterial smooth muscle responses to hypoxia.Novel Flurometric Tool to Assess Mitochondrial Redox State of Isolated Perfused Rat Lungs after Exposure to Hyperoxia.Potentiation of the hypoxic ventilatory response by 1 day of hyperoxia in neonatal rats.Prevention of hyperoxia-induced bronchial hyperreactivity by sildenafil and vasoactive intestinal peptide: impact of preserved lung function and structure Two-photon imaging within the murine thorax without respiratory and cardiac motion artifact.Differential responses of targeted lung redox enzymes to rat exposure to 60 or 85% oxygen.In vivo detection of hyperoxia-induced pulmonary endothelial cell death using (99m)Tc-duramycin.Differential lung uptake of 99mTc-hexamethylpropyleneamine oxime and 99mTc-duramycin in the chronic hyperoxia rat model.Lung injury caused by high tidal volume mechanical ventilation and hyperoxia is dependent on oxidant-mediated c-Jun NH2-terminal kinase activation.High level of oxygen treatment causes cardiotoxicity with arrhythmias and redox modulation Hyperoxia activates ATM independent from mitochondrial ROS and dysfunction.NADPH oxidase-derived ROS and the regulation of pulmonary vessel tone Prevention of lung injury in cardiac surgery: a review Hyperoxia promotes polarization of the immune response in ovalbumin-induced airway inflammation, leading to a TH17 cell phenotype Role of glutathione in lung retention of 99mTc-hexamethylpropyleneamine oxime in two unique rat models of hyperoxic lung injury Superoxide dismutase improves oxygenation and reduces oxidation in neonatal pulmonary hypertension.Real-time lung microscopy.Mitochondrial reactive oxygen species-mediated signaling in endothelial cells.Inhibition of the phospholipase A2 activity of peroxiredoxin 6 prevents lung damage with exposure to hyperoxia.Endothelial cell respiration is affected by the oxygen tension during shear exposure: role of mitochondrial peroxynitrite.Hypoxic stress exacerbates hyperoxia-induced lung injury in a neonatal mouse model of bronchopulmonary dysplasia Coenzyme Q1 redox metabolism during passage through the rat pulmonary circulation and the effect of hyperoxia 20-HETE increases survival and decreases apoptosis in pulmonary arteries and pulmonary artery endothelial cells Phospholipase D-mediated activation of IQGAP1 through Rac1 regulates hyperoxia-induced p47phox translocation and reactive oxygen species generation in lung endothelial cells.Manipulation of gene expression by oxygen: a primer from bedside to bench Stress response of the trabecular meshwork.NOX enzymes: potential target for the treatment of acute lung injury.Hyperoxia: a review of the risks and benefits in adult cardiac surgery.Endothelial cell regulation of pulmonary vascular tone, inflammation, and coagulation.Cardiovascular effects of hyperoxia during and after cardiac surgery.

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P2860

Hyperoxia-induced reactive oxygen species formation in pulmonary capillary endothelial cells in situ.

http://www.wikidata.org/entity/Q46853361

description

2005 nî lūn-bûn

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2005年の論文

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2005年学术文章

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2005年学术文章

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2005年学术文章

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2005年学术文章

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2005年学术文章

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2005年学术文章

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2005年學術文章

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2005年學術文章

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name

Hyperoxia-induced reactive oxy ...... ary endothelial cells in situ.

@en

Hyperoxia-induced reactive oxy ...... ary endothelial cells in situ.

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type

label

Hyperoxia-induced reactive oxy ...... ary endothelial cells in situ.

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Hyperoxia-induced reactive oxy ...... ary endothelial cells in situ.

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prefLabel

Hyperoxia-induced reactive oxy ...... ary endothelial cells in situ.

@en

Hyperoxia-induced reactive oxy ...... ary endothelial cells in situ.

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RCMB.2005-0223OC

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American Journal of Respiratory Cell and Molecular Biology

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Hyperoxia-induced reactive oxy ...... ary endothelial cells in situ.

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P2093

Alexander Kerem

http://www.w3.org/2001/XMLSchema#string

Corinna Brueckl

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Fritz Krombach

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Helmut Habazettl

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Hermann Kuppe

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Stephanie Kaestle

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Wolfgang M Kuebler

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P2860

Dual oxidase-2 has an intrinsic Ca2+-dependent H2O2-generating activity

Mitochondrial formation of reactive oxygen species

Differential contribution of various adhesion molecules to leukocyte kinetics in pulmonary microvessels of hyperoxia-exposed rat lungs.

Redox regulation of signal transduction in cardiac and smooth muscle.

Pressure is proinflammatory in lung venular capillaries.

Detection and characterization of the product of hydroethidine and intracellular superoxide by HPLC and limitations of fluorescence

Mitochondrial dysfunction in cardiac disease: ischemia--reperfusion, aging, and heart failure.

Mice lacking extracellular superoxide dismutase are more sensitive to hyperoxia.

Transgenic mice with expression of elevated levels of copper-zinc superoxide dismutase in the lungs are resistant to pulmonary oxygen toxicity.

Mechanism of Ca2+ activation of the NADPH oxidase 5 (NOX5).

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453-463

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10.1165/RCMB.2005-0223OC

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4

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34

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16357365

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