Is Nox4 a key regulator of the activated state of fibroblasts in systemic sclerosis?
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Computational Modeling Predicts Simultaneous Targeting of Fibroblasts and Epithelial Cells Is Necessary for Treatment of Pulmonary Fibrosis.Targeting of NADPH oxidase in vitro and in vivo suppresses fibroblast activation and experimental skin fibrosis.Therapeutic potential of NADPH oxidase 1/4 inhibitors.A reactive oxygen species-mediated loop maintains increased expression of NADPH oxidases 2 and 4 in skin fibroblasts from patients with systemic sclerosis.
P2860
Is Nox4 a key regulator of the activated state of fibroblasts in systemic sclerosis?
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Is Nox4 a key regulator of the activated state of fibroblasts in systemic sclerosis?
@en
Is Nox4 a key regulator of the activated state of fibroblasts in systemic sclerosis?
@nl
type
label
Is Nox4 a key regulator of the activated state of fibroblasts in systemic sclerosis?
@en
Is Nox4 a key regulator of the activated state of fibroblasts in systemic sclerosis?
@nl
prefLabel
Is Nox4 a key regulator of the activated state of fibroblasts in systemic sclerosis?
@en
Is Nox4 a key regulator of the activated state of fibroblasts in systemic sclerosis?
@nl
P2093
P2860
P356
P1476
Is Nox4 a key regulator of the activated state of fibroblasts in systemic sclerosis?
@en
P2093
Claus Kerkhoff
Heba Dosoki
Markus Böhm
P2860
P304
P356
10.1111/EXD.12497
P577
2014-09-01T00:00:00Z