Targeted degradation of c-Fos, but not v-Fos, by a phosphorylation-dependent signal on c-Jun.
about
Fanconi anemia proteins FANCA, FANCC, and FANCG/XRCC9 interact in a functional nuclear complexInteraction of elicitor-induced DNA-binding proteins with elicitor response elements in the promoters of parsley PR1 genesMammalian Sug1 and c-Fos in the nuclear 26S proteasomeProteasome complex as a potential cellular target of hepatitis B virus X proteinThe N terminus of microsomal delta 9 stearoyl-CoA desaturase contains the sequence determinant for its rapid degradationIdentification of in vivo phosphorylation sites of SET, a nuclear phosphoprotein encoded by the translocation breakpoint in acute undifferentiated leukemiaGeneration of destabilized green fluorescent protein as a transcription reporterThe Mos/MAP kinase pathway stabilizes c-Fos by phosphorylation and augments its transforming activity in NIH 3T3 cellsGonadotropin-releasing hormone pulse sensitivity of follicle-stimulating hormone-beta gene is mediated by differential expression of positive regulatory activator protein 1 factors and corepressors SKIL and TGIF1.Identification of protein instability determinants in the carboxy-terminal region of c-Myb removed as a result of retroviral integration in murine monocytic leukemias.Destruction of Myc by ubiquitin-mediated proteolysis: cancer-associated and transforming mutations stabilize Myc.Marek's disease virus Meq transforms chicken cells via the v-Jun transcriptional cascade: a converging transforming pathway for avian oncoviruses.Degradation of the proto-oncogene product c-Fos by the ubiquitin proteolytic system in vivo and in vitro: identification and characterization of the conjugating enzymes.The carboxy-terminal transactivation domain of Oct-4 acquires cell specificity through the POU domain.Role of 26S proteasome and HRD genes in the degradation of 3-hydroxy-3-methylglutaryl-CoA reductase, an integral endoplasmic reticulum membrane protein.Multiple regulatory elements in the interleukin-6 gene mediate induction by prostaglandins, cyclic AMP, and lipopolysaccharide.Prolonged α-amanitin treatment of cells for studying mutated polymerases causes degradation of DSIF160 and other proteins.Ubiquitination and degradation of ATF2 are dimerization dependent.Complex functions of AP-1 transcription factors in differentiation and survival of PC12 cells.Chaperone Hsp27 modulates AUF1 proteolysis and AU-rich element-mediated mRNA degradation.c-Jun activation is required for 4-hydroxytamoxifen-induced cell death in breast cancer cells.Antioxidant-induced changes of the AP-1 transcription complex are paralleled by a selective suppression of human papillomavirus transcription.Degradation of c-Fos by the 26S proteasome is accelerated by c-Jun and multiple protein kinases.Hsp27 and F-box protein β-TrCP promote degradation of mRNA decay factor AUF1.Ubiquitination of the G1 cyclin Cln2p by a Cdc34p-dependent pathway.Down-regulation of the neurotrophin receptor TrkB following ligand binding. Evidence for an involvement of the proteasome and differential regulation of TrkA and TrkB.Epidermal growth factor and okadaic acid stimulate Sp1 proteolysis.Multisite phosphorylation of ornithine decarboxylase in transformed macrophages results in increased intracellular enzyme stability and catalytic efficiency.Transcription factors in normal and neoplastic pituitary tissues.Involvement of the proteasome and antizyme in ornithine decarboxylase degradation by a reticulocyte lysate.Cell transformation by c-fos requires an extended period of expression and is independent of the cell cycle.c-Jun homodimers can function as a context-specific coactivator.c-Fos proteasomal degradation is activated by a default mechanism, and its regulation by NAD(P)H:quinone oxidoreductase 1 determines c-Fos serum response kinetics.Degradation of proto-oncoprotein c-Rel by the ubiquitin-proteasome pathway.PEST motifs are not required for rapid calpain-mediated proteolysis of c-fos protein.Pti4 is induced by ethylene and salicylic acid, and its product is phosphorylated by the Pto kinase.Conversion of HPV 18 positive non-tumorigenic HeLa-fibroblast hybrids to invasive growth involves loss of TNF-alpha mediated repression of viral transcription and modification of the AP-1 transcription complex.
P2860
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P2860
Targeted degradation of c-Fos, but not v-Fos, by a phosphorylation-dependent signal on c-Jun.
description
1992 nî lūn-bûn
@nan
1992年の論文
@ja
1992年学术文章
@wuu
1992年学术文章
@zh
1992年学术文章
@zh-cn
1992年学术文章
@zh-hans
1992年学术文章
@zh-my
1992年学术文章
@zh-sg
1992年學術文章
@yue
1992年學術文章
@zh-hant
name
Targeted degradation of c-Fos, ...... ion-dependent signal on c-Jun.
@en
Targeted degradation of c-Fos, ...... ion-dependent signal on c-Jun.
@nl
type
label
Targeted degradation of c-Fos, ...... ion-dependent signal on c-Jun.
@en
Targeted degradation of c-Fos, ...... ion-dependent signal on c-Jun.
@nl
prefLabel
Targeted degradation of c-Fos, ...... ion-dependent signal on c-Jun.
@en
Targeted degradation of c-Fos, ...... ion-dependent signal on c-Jun.
@nl
P2093
P356
P1433
P1476
Targeted degradation of c-Fos, ...... ion-dependent signal on c-Jun.
@en
P2093
Chavrier C
Papavassiliou AG
P304
P356
10.1126/SCIENCE.1470918
P407
P577
1992-12-01T00:00:00Z