Early growth response-1 contributes to galactosamine/lipopolysaccharide-induced acute liver injury in mice.
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Kupffer cells in the liverFarnesyltransferase inhibitor improved survival following endotoxin challenge in mice.Hepatic fibrosis is enhanced and accompanied by robust oval cell activation after chronic carbon tetrachloride administration to Egr-1-deficient miceExogenous thioredoxin prevents ethanol-induced oxidative damage and apoptosis in mouse liver.Moderate, chronic ethanol feeding exacerbates carbon-tetrachloride-induced hepatic fibrosis via hepatocyte-specific hypoxia inducible factor 1αEarly growth response-1 attenuates liver injury and promotes hepatoprotection after carbon tetrachloride exposure in miceBile acids induce inflammatory genes in hepatocytes: a novel mechanism of inflammation during obstructive cholestasis.Alternative complement pathway component Factor D contributes to efficient clearance of tissue debris following acute CCl₄-induced injury.Early growth response (EGR)-1 is required for timely cell-cycle entry and progression in hepatocytes after acute carbon tetrachloride exposure in mice.Complement and alcoholic liver disease: role of C1q in the pathogenesis of ethanol-induced liver injury in miceEarly growth response factor-1 limits biliary fibrosis in a model of xenobiotic-induced cholestasis in mice.Inhibition of apoptosis protects mice from ethanol-mediated acceleration of early markers of CCl4 -induced fibrosis but not steatosis or inflammation.An overview of animal models for investigating the pathogenesis and therapeutic strategies in acute hepatic failure.Gene expression signatures of peripheral CD4+ T cells clearly discriminate between patients with acute and chronic hepatitis B infection.Myeloid-MyD88 Contributes to Ethanol-Induced Liver Injury in Mice Linking Hepatocellular Death to Inflammation.Protective effect of carbon monoxide pre-conditioning on LPS-induced endothelial cell stress.Deletion of caveolin-1 attenuates LPS/GalN-induced acute liver injury in mice
P2860
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P2860
Early growth response-1 contributes to galactosamine/lipopolysaccharide-induced acute liver injury in mice.
description
2007 nî lūn-bûn
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2007年の論文
@ja
2007年学术文章
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2007年学术文章
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2007年学术文章
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2007年学术文章
@zh-hans
2007年学术文章
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2007年学术文章
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2007年學術文章
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2007年學術文章
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name
Early growth response-1 contri ...... ed acute liver injury in mice.
@en
Early growth response-1 contri ...... ed acute liver injury in mice.
@nl
type
label
Early growth response-1 contri ...... ed acute liver injury in mice.
@en
Early growth response-1 contri ...... ed acute liver injury in mice.
@nl
prefLabel
Early growth response-1 contri ...... ed acute liver injury in mice.
@en
Early growth response-1 contri ...... ed acute liver injury in mice.
@nl
P2093
P2860
P356
P1476
Early growth response-1 contri ...... ed acute liver injury in mice.
@en
P2093
Gavin E Arteel
Laura E Nagy
Luping Guo
Megan R McMullen
Michele T Pritchard
Sanjoy Roychowdhury
P2860
P304
P356
10.1152/AJPGI.00325.2007
P577
2007-10-04T00:00:00Z