Bradykinin prevents reperfusion injury by targeting mitochondrial permeability transition pore through glycogen synthase kinase 3beta.
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Myocardial autophagic energy stress responses--macroautophagy, mitophagy, and glycophagyIntramitochondrial signaling: interactions among mitoKATP, PKCepsilon, ROS, and MPTInvolvment of cytosolic and mitochondrial GSK-3beta in mitochondrial dysfunction and neuronal cell death of MPTP/MPP-treated neuronsBradykinin and adenosine receptors mediate desflurane induced postconditioning in human myocardium: role of reactive oxygen species.GSK3beta promotes apoptosis after renal ischemic injury.Pharmacological postconditioning treatment of myocardial infarction with netrin-1.Mitochondrial integrity: preservation through Akt/Pim-1 kinase signaling in the cardiomyocyteMechanism for resveratrol-induced cardioprotection against reperfusion injury involves glycogen synthase kinase 3beta and mitochondrial permeability transition pore.Effects of postconditioning with N,N,N'N'-tetrakis-[2-pyridylmethyl]-ethylenediamine in isolated rat hearts.Post-conditioning with gradually increased reperfusion provides better cardioprotection in rats.AMPK-Dependent Metabolic Regulation by PPAR Agonists.Mitigation of carbon tetrachloride-induced hepatic injury by methylene blue, a repurposed drug, is mediated by dual inhibition of GSK3β downstream of PKA.Clusterin protects H9c2 cardiomyocytes from oxidative stress-induced apoptosis via Akt/GSK-3β signaling pathway.Sevoflurane postconditioning protects rat hearts against ischemia-reperfusion injury via the activation of PI3K/AKT/mTOR signalingGinsenoside Rd attenuates myocardial ischemia/reperfusion injury via Akt/GSK-3β signaling and inhibition of the mitochondria-dependent apoptotic pathwayMorphine-induced postconditioning modulates mitochondrial permeability transition pore opening via delta-1 opioid receptors activation in isolated rat hearts.Bradykinin B1 and B2 receptors both have protective roles in renal ischemia/reperfusion injury.Differential role of kinin B1 and B2 receptors in ischemia-induced apoptosis and ventricular remodelingAstragaloside IV inhibits oxidative stress-induced mitochondrial permeability transition pore opening by inactivating GSK-3β via nitric oxide in H9c2 cardiac cells.Cardiac inflammation after local irradiation is influenced by the kallikrein-kinin systemGSK-3β downregulates Nrf2 in cultured cortical neurons and in a rat model of cerebral ischemia-reperfusion.IB-MECA and cardioprotection.Inhibition of glycogen synthase kinase 3beta prevents peroxide-induced collapse of mitochondrial membrane potential in rat ventricular myocytes.Exogenous zinc protects cardiac cells from reperfusion injury by targeting mitochondrial permeability transition pore through inactivation of glycogen synthase kinase-3beta.Conditioning the heart induces formation of signalosomes that interact with mitochondria to open mitoKATP channelsAkt activation improves oxidative phosphorylation in renal proximal tubular cells following nephrotoxicant injuryPharmacologic therapeutics for cardiac reperfusion injury.The kallikrein-kinin system in health and in diseases of the kidney.Regulation of hexokinase binding to VDACRole of glycogen synthase kinase-3beta in cardioprotection.Adenosine A2A and A2B receptors work in concert to induce a strong protection against reperfusion injury in rat hearts.Tauroursodeoxycholic acid inhibits endoplasmic reticulum stress, blocks mitochondrial permeability transition pore opening, and suppresses reperfusion injury through GSK-3ß in cardiac H9c2 cells.The potential dual effects of sevoflurane on AKT/GSK3β signaling pathwayAssociation of glycogen synthase kinase-3β with Parkinson's disease (review).Novel perspectives on the PHD-HIF oxygen sensing pathway in cardioprotection mediated by IPC and RIPCRole of connexin-43 in protective PI3K-Akt-GSK-3β signaling in cardiomyocytes.Glycogen Synthase Kinase 3β Influences Injury Following Cerebral Ischemia/Reperfusion in Rats.Neuroprotective effect of kinin B1 receptor activation in acute cerebral ischemia in diabetic mice.Adenosine receptors: regulatory players in the preservation of mitochondrial function induced by ischemic preconditioning of rat liver.AMPK-associated signaling to bridge the gap between fuel metabolism and hepatocyte viability.
P2860
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P2860
Bradykinin prevents reperfusion injury by targeting mitochondrial permeability transition pore through glycogen synthase kinase 3beta.
description
2006 nî lūn-bûn
@nan
2006年の論文
@ja
2006年学术文章
@wuu
2006年学术文章
@zh
2006年学术文章
@zh-cn
2006年学术文章
@zh-hans
2006年学术文章
@zh-my
2006年学术文章
@zh-sg
2006年學術文章
@yue
2006年學術文章
@zh-hant
name
Bradykinin prevents reperfusio ...... lycogen synthase kinase 3beta.
@en
Bradykinin prevents reperfusio ...... lycogen synthase kinase 3beta.
@nl
type
label
Bradykinin prevents reperfusio ...... lycogen synthase kinase 3beta.
@en
Bradykinin prevents reperfusio ...... lycogen synthase kinase 3beta.
@nl
prefLabel
Bradykinin prevents reperfusio ...... lycogen synthase kinase 3beta.
@en
Bradykinin prevents reperfusio ...... lycogen synthase kinase 3beta.
@nl
P2093
P1476
Bradykinin prevents reperfusio ...... lycogen synthase kinase 3beta.
@en
P2093
Robert A Mueller
Sung-Sik Park
Zhelong Xu
P304
P356
10.1016/J.YJMCC.2006.01.024
P577
2006-03-06T00:00:00Z