about
Inactivation of the HIF-1α/PDK3 signaling axis drives melanoma toward mitochondrial oxidative metabolism and potentiates the therapeutic activity of pro-oxidantsTargeting chelatable iron as a therapeutic modality in Parkinson's disease.Exploiting mitochondrial dysfunction for effective elimination of imatinib-resistant leukemic cells.Mitochondrial oxidative stress is the Achille's heel of melanoma cells resistant to Braf-mutant inhibitor.Mitochondrial oxidative phosphorylation controls cancer cell's life and death decisions upon exposure to MAPK inhibitors.Another facet to the anticancer response to lamellarin D: induction of cellular senescence through inhibition of topoisomerase I and intracellular Ros productionGILZ overexpression attenuates endoplasmic reticulum stress-mediated cell death via the activation of mitochondrial oxidative phosphorylation.Metabolic rewiring in cancer cells overexpressing the glucocorticoid-induced leucine zipper protein (GILZ): Activation of mitochondrial oxidative phosphorylation and sensitization to oxidative cell death induced by mitochondrial targeted drugs.Regulation by survivin of cancer cell death induced by F14512, a polyamine-containing inhibitor of DNA topoisomerase II.Inhibition of mitochondrial respiration mediates apoptosis induced by the anti-tumoral alkaloid lamellarin D.Essential role of mitochondria in apoptosis of cancer cells induced by the marine alkaloid Lamellarin D.
P50
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P50
name
Jérome Kluza
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Jérome Kluza
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Jérome Kluza
@es
Jérome Kluza
@fr
Jérome Kluza
@nl
Jérome Kluza
@sl
type
label
Jérome Kluza
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Jérome Kluza
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Jérome Kluza
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Jérome Kluza
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Jérome Kluza
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Jérome Kluza
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prefLabel
Jérome Kluza
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Jérome Kluza
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Jérome Kluza
@es
Jérome Kluza
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Jérome Kluza
@nl
Jérome Kluza
@sl
P214
P269
P1412
P21
P214
P269
P27
P31
P7859
viaf-313562680