Chronic activation of dorsal hindbrain corticosteroid receptors augments the arterial pressure response to acute stress.
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Glucocorticoid receptors in the nucleus of the solitary tract (NTS) decrease endocrine and behavioral stress responsesElevated corticosterone in the dorsal hindbrain increases plasma norepinephrine and neuropeptide Y, and recruits a vasopressin response to stressChronic blockade of hindbrain glucocorticoid receptors reduces blood pressure responses to novel stress and attenuates adaptation to repeated stressKnockdown of tyrosine hydroxylase in the nucleus of the solitary tract reduces elevated blood pressure during chronic intermittent hypoxia.Changes in C57BL6 Mouse Hippocampal Transcriptome Induced by Hypergravity Mimic Acute Corticosterone-Induced StressRegulation of the stress response in rats by central actions of glucocorticoids.Ascending mechanisms of stress integration: Implications for brainstem regulation of neuroendocrine and behavioral stress responses.The relationship of stress and blood pressure effectors.
P2860
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P2860
Chronic activation of dorsal hindbrain corticosteroid receptors augments the arterial pressure response to acute stress.
description
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name
Chronic activation of dorsal h ...... sure response to acute stress.
@en
Chronic activation of dorsal h ...... sure response to acute stress.
@nl
type
label
Chronic activation of dorsal h ...... sure response to acute stress.
@en
Chronic activation of dorsal h ...... sure response to acute stress.
@nl
prefLabel
Chronic activation of dorsal h ...... sure response to acute stress.
@en
Chronic activation of dorsal h ...... sure response to acute stress.
@nl
P2093
P2860
P1433
P1476
Chronic activation of dorsal h ...... sure response to acute stress.
@en
P2093
Andrea G Bechtold
Deborah A Scheuer
Kathy A Vernon
P2860
P304
P356
10.1161/01.HYP.0000250088.15021.C2
P407
P577
2006-11-06T00:00:00Z