Cardiovascular phenotype in Smad3 deficient mice with renovascular hypertension.

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Ccl2 deficiency protects against chronic renal injury in murine renovascular hypertension.

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Q55276177-0E67FBBE-FF3C-4C9E-8FF0-E6EBBE9332BF

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Cardiovascular phenotype in Smad3 deficient mice with renovascular hypertension.

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2017 nî lūn-bûn

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2017年学术文章

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2017年学术文章

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Cardiovascular phenotype in Smad3 deficient mice with renovascular hypertension.

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Cardiovascular phenotype in Smad3 deficient mice with renovascular hypertension.

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Item

label

Cardiovascular phenotype in Smad3 deficient mice with renovascular hypertension.

@en

Cardiovascular phenotype in Smad3 deficient mice with renovascular hypertension.

@nl

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Cardiovascular phenotype in Smad3 deficient mice with renovascular hypertension.

@en

Cardiovascular phenotype in Smad3 deficient mice with renovascular hypertension.

@nl

P1476

Cardiovascular phenotype in Smad3 deficient mice with renovascular hypertension.

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P2093

Feng Gao

http://www.w3.org/2001/XMLSchema#string

Gina Warner

http://www.w3.org/2001/XMLSchema#string

Karen R Lien

http://www.w3.org/2001/XMLSchema#string

Karl Nath

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Mazen Osman

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Sonu Kashyap

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Yousif Al Saiegh

http://www.w3.org/2001/XMLSchema#string

Zeng Hu

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P2860

SMAD3 deficiency promotes inflammatory aortic aneurysms in angiotensin II-infused mice via activation of iNOS

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Aneurysm syndromes caused by mutations in the TGF-beta receptor

Smads: transcriptional activators of TGF-beta responses

Stenting and medical therapy for atherosclerotic renal-artery stenosis

Inhibition of transforming growth factor-beta signaling induces left ventricular dilation and dysfunction in the pressure-overloaded heart

Transforming growth factor-beta signal transduction and progressive renal disease.

Exome sequencing identifies SMAD3 mutations as a cause of familial thoracic aortic aneurysm and dissection with intracranial and other arterial aneurysms.

Ischaemic nephropathy secondary to atherosclerotic renal artery stenosis: clinical and histopathological correlates.

Transforming growth factor-beta signaling through the Smad pathway: role in extracellular matrix gene expression and regulation.

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e0187062

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scholarly article

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10.1371/JOURNAL.PONE.0187062

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English

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P478

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Joseph P Grande

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2017-10-26T00:00:00Z

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29073282

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5658153

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Cardiovascular phenotype in Smad3 deficient mice with renovascular hypertension.

about

P2860

P2860

Cardiovascular phenotype in Smad3 deficient mice with renovascular hypertension.

description

name

type

label

prefLabel

P1433

P1476

P2093

P2860

P304

P31

P356

P407

P433

P478

P50

P577

P698

P932

P356

P1433

P1476

P2093

P2860

P304

P31

P356

P407

P433

P478

P50

P577

P698

P932