PI3Kδ activates E2F1 synthesis in response to mRNA translation stress.

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EBNA1: Oncogenic Activity, Immune Evasion and Biochemical Functions Provide Targets for Novel Therapeutic Strategies against Epstein-Barr Virus- Associated Cancers.Lymphomas driven by Epstein-Barr virus nuclear antigen-1 (EBNA1) are dependant upon Mdm2.

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PI3Kδ activates E2F1 synthesis in response to mRNA translation stress.

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PI3Kδ activates E2F1 synthesis in response to mRNA translation stress.

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PI3Kδ activates E2F1 synthesis in response to mRNA translation stress.

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PI3Kδ activates E2F1 synthesis in response to mRNA translation stress.

@en

PI3Kδ activates E2F1 synthesis in response to mRNA translation stress.

@nl

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PI3Kδ activates E2F1 synthesis in response to mRNA translation stress.

@en

PI3Kδ activates E2F1 synthesis in response to mRNA translation stress.

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PI3Kδ activates E2F1 synthesis in response to mRNA translation stress.

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Chrysoula Daskalogianni

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Joanna B Wilson

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Karin Nylander

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Robin Fåhraeus

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Slovénie Pyndiah

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Inactivation of PI(3)K p110δ breaks regulatory T-cell-mediated immune tolerance to cancer

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Molecular mechanisms of mTOR-mediated translational control

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s41467-017-02282-w

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2103

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scholarly article

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10.1038/S41467-017-02282-W

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English

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Sivakumar Vadivel Gnanasundram

Kate Armfield

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2017-12-13T00:00:00Z

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29235459

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5727396

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PI3Kδ activates E2F1 synthesis in response to mRNA translation stress.

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P2860

P2860

PI3Kδ activates E2F1 synthesis in response to mRNA translation stress.

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P1433

P1476

P2093

P2860

P2888

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P31

P356

P407

P433

P478

P50

P577

P698

P932

P356

P1433

P1476

P2093

P2860

P2888

P304

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P356

P407

P433

P478

P50

P577

P698

P932