Ascorbic acid promotes 3T3-L1 cells adipogenesis by attenuating ERK signaling to upregulate the collagen VI.

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Ascorbic acid promotes 3T3-L1 cells adipogenesis by attenuating ERK signaling to upregulate the collagen VI.

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2017 nî lūn-bûn

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2017年の論文

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2017年学术文章

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Ascorbic acid promotes 3T3-L1 ...... to upregulate the collagen VI.

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Ascorbic acid promotes 3T3-L1 ...... to upregulate the collagen VI.

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Ascorbic acid promotes 3T3-L1 ...... to upregulate the collagen VI.

@en

Ascorbic acid promotes 3T3-L1 ...... to upregulate the collagen VI.

@nl

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Ascorbic acid promotes 3T3-L1 ...... to upregulate the collagen VI.

@en

Ascorbic acid promotes 3T3-L1 ...... to upregulate the collagen VI.

@nl

P1476

Ascorbic acid promotes 3T3-L1 ...... to upregulate the collagen VI.

@en

P2093

Chang Liu

http://www.w3.org/2001/XMLSchema#string

Chuanguo Liu

http://www.w3.org/2001/XMLSchema#string

Congcong Guo

http://www.w3.org/2001/XMLSchema#string

Guorong Li

http://www.w3.org/2001/XMLSchema#string

Jie Pan

http://www.w3.org/2001/XMLSchema#string

Kun Huang

http://www.w3.org/2001/XMLSchema#string

Pingping Wang

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Zongguo Sun

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P2860

A plasma kallikrein-dependent plasminogen cascade required for adipocyte differentiation

Transcription factor Ets-1 is essential for mesangial matrix remodeling

Mitotic clonal expansion: a synchronous process required for adipogenesis

Ascorbic acid enhances differentiation of embryonic stem cells into cardiac myocytes.

Adipocyte-derived collagen VI affects early mammary tumor progression in vivo, demonstrating a critical interaction in the tumor/stroma microenvironment

Role of pathways for signal transducers and activators of transcription, and mitogen-activated protein kinase in adipocyte differentiation.

CCAAT/enhancer-binding protein beta is required for mitotic clonal expansion during adipogenesis.

Protein hydroxylation: prolyl 4-hydroxylase, an enzyme with four cosubstrates and a multifunctional subunit.

Regulatory mechanisms of mitogen-activated kinase signaling.

Regulation of collagen synthesis by ascorbic acid.

P2888

s12986-017-0234-y

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scholarly article

P356

10.1186/S12986-017-0234-Y

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P478

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P577

2017-12-28T00:00:00Z

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1100087148

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29299041

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5745638

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