DEK promoted EMT and angiogenesis through regulating PI3K/AKT/mTOR pathway in triple-negative breast cancer.

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miR-3178 inhibits cell proliferation and metastasis by targeting Notch1 in triple-negative breast cancer The "Yin and Yang" of Natural Compounds in Anticancer Therapy of Triple-Negative Breast Cancers

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DEK promoted EMT and angiogenesis through regulating PI3K/AKT/mTOR pathway in triple-negative breast cancer.

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2017 nî lūn-bûn

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2017年の論文

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2017年学术文章

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2017年学术文章

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2017年学术文章

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2017年学术文章

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2017年学术文章

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2017年学术文章

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2017年學術文章

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2017年學術文章

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name

DEK promoted EMT and angiogene ...... triple-negative breast cancer.

@en

DEK promoted EMT and angiogene ...... triple-negative breast cancer.

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type

Item

label

DEK promoted EMT and angiogene ...... triple-negative breast cancer.

@en

DEK promoted EMT and angiogene ...... triple-negative breast cancer.

@nl

prefLabel

DEK promoted EMT and angiogene ...... triple-negative breast cancer.

@en

DEK promoted EMT and angiogene ...... triple-negative breast cancer.

@nl

P1476

DEK promoted EMT and angiogene ...... triple-negative breast cancer.

@en

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Guang Zhu

http://www.w3.org/2001/XMLSchema#string

Liyan Chen

http://www.w3.org/2001/XMLSchema#string

Meihua Gao

http://www.w3.org/2001/XMLSchema#string

Tiefeng Jin

http://www.w3.org/2001/XMLSchema#string

Yang Yang

http://www.w3.org/2001/XMLSchema#string

Yixuan Wang

http://www.w3.org/2001/XMLSchema#string

Yu Jin

http://www.w3.org/2001/XMLSchema#string

Zhenhua Lin

http://www.w3.org/2001/XMLSchema#string

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The human DEK oncogene regulates DNA damage response signaling and repair

The protein encoded by the proto-oncogene DEK changes the topology of chromatin and reduces the efficiency of DNA replication in a chromatin-specific manner

The translocation (6;9), associated with a specific subtype of acute myeloid leukemia, results in the fusion of two genes, dek and can, and the expression of a chimeric, leukemia-specific dek-can mRNA

The acute myeloid leukemia-associated protein, DEK, forms a splicing-dependent interaction with exon-product complexes

PI3K/PTEN signaling in angiogenesis and tumorigenesis

Forced expression of the DEK-NUP214 fusion protein promotes proliferation dependent on upregulation of mTOR

Targeting PI3K signalling in cancer: opportunities, challenges and limitations

The Scribble polarity protein stabilizes E-cadherin/p120-catenin binding and blocks retrieval of E-cadherin to the Golgi.

Active kinase profiling, genetic and pharmacological data define mTOR as an important common target in triple-negative breast cancer.

DEK associates with tumor stage and outcome in HPV16 positive oropharyngeal squamous cell carcinoma.

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98708-98722

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scholarly article

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10.18632/ONCOTARGET.21864

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English

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2017-10-17T00:00:00Z

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29228721

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P932

5716761

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DEK promoted EMT and angiogenesis through regulating PI3K/AKT/mTOR pathway in triple-negative breast cancer.

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P2860

DEK promoted EMT and angiogenesis through regulating PI3K/AKT/mTOR pathway in triple-negative breast cancer.

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