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MicroRNAs: novel regulators involved in the pathogenesis of psoriasis?Transition from inflammation to proliferation: a critical step during wound healingMicroRNAs and immunity: novel players in the regulation of normal immune function and inflammationThe expression of microRNA-203 during human skin morphogenesisAntioxidants protect keratinocytes against M. ulcerans mycolactone cytotoxicityA genome-wide association study identifies new psoriasis susceptibility loci and an interaction between HLA-C and ERAP1.Evaluation of an interactive case simulation system in dermatology and venereology for medical studentsGenetic polymorphisms altering microRNA activity in psoriasis--a key to solve the puzzle of missing heritability?CD49a Expression Defines Tissue-Resident CD8+ T Cells Poised for Cytotoxic Function in Human Skin.HLA-Cw*0602 associates with a twofold higher prevalence of positive streptococcal throat swab at the onset of psoriasis: a case control studyMiR-155 is overexpressed in patients with atopic dermatitis and modulates T-cell proliferative responses by targeting cytotoxic T lymphocyte-associated antigen 4.Accelerating translational research by clinically driven development of an informatics platform--a case study.Low salivary dehydroepiandrosterone and androgen-regulated cysteine-rich secretory protein 3 levels in Sjögren's syndrome.LC-MS metabolomics of psoriasis patients reveals disease severity-dependent increases in circulating amino acids that are ameliorated by anti-TNFα treatment.Protein kinase C-dependent upregulation of miR-203 induces the differentiation of human keratinocytes.MicroRNA-31 is overexpressed in cutaneous squamous cell carcinoma and regulates cell motility and colony formation ability of tumor cells.Genome-wide meta-analysis identifies multiple novel associations and ethnic heterogeneity of psoriasis susceptibility.Circulating levels of sphingosine-1-phosphate are elevated in severe, but not mild psoriasis and are unresponsive to anti-TNF-α treatment.Neutrophils and keratinocytes in innate immunity--cooperative actions to provide antimicrobial defense at the right time and place.MicroRNA-132 enhances transition from inflammation to proliferation during wound healing.MicroRNA-125b down-regulates matrix metallopeptidase 13 and inhibits cutaneous squamous cell carcinoma cell proliferation, migration, and invasionGenetic association with ERAP1 in psoriasis is confined to disease onset after puberty and not dependent on HLA-C*06.Metabolic disorders in patients with psoriasis and psoriatic arthritis.RNA editing of the GLI1 transcription factor modulates the output of Hedgehog signalingHuman antimicrobial protein hCAP18/LL-37 promotes a metastatic phenotype in breast cancer.Psoriasis and systemic inflammatory diseases: potential mechanistic links between skin disease and co-morbid conditions.Effect of Ixekizumab Treatment on Work Productivity for Patients With Moderate-to-Severe Plaque Psoriasis: Analysis of Results From 3 Randomized Phase 3 Clinical Trials.Epidermal Th22 and Tc17 cells form a localized disease memory in clinically healed psoriasis.Sebocytes differentially express and secrete adipokines.Economic Burden of Psoriasis and Potential Cost Offsets with Biologic Treatment: A Swedish Register Analysis.HLA-C expression pattern is spatially different between psoriasis and eczema skin lesions.Antimicrobial protein hCAP18/LL-37 is highly expressed in breast cancer and is a putative growth factor for epithelial cells.STG does not associate with psoriasis in the Swedish population.Psoriasis Skin Inflammation-Induced microRNA-26b Targets NCEH1 in Underlying Subcutaneous Adipose Tissue.Increased cause-specific mortality in patients with mild and severe psoriasis: a population-based Swedish register study.MicroRNA-132 promotes fibroblast migration via regulating RAS p21 protein activator 1 in skin wound healing.Interleukin-8 is regulated by miR-203 at the posttranscriptional level in primary human keratinocytes.MicroRNA-31 is overexpressed in psoriasis and modulates inflammatory cytokine and chemokine production in keratinocytes via targeting serine/threonine kinase 40.Treatment with LL-37 is safe and effective in enhancing healing of hard-to-heal venous leg ulcers: a randomized, placebo-controlled clinical trial.Topical treatment with the vitamin D analogue calcipotriol enhances the upregulation of the antimicrobial protein hCAP18/LL-37 during wounding in human skin in vivo.
P50
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