Hydrogen Sulfide Reduces Recruitment of CD11b+Gr-1+ Cells in Mice With Myocardial Infarction.

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The Role of Hydrogen Sulfide on Cardiovascular Homeostasis: An Overview with Update on Immunomodulation An Update on Hydrogen Sulfide and Nitric Oxide Interactions in the Cardiovascular System Hydrogen Sulfide (HS)-Releasing Compounds: Therapeutic Potential in Cardiovascular Diseases

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Hydrogen Sulfide Reduces Recruitment of CD11b+Gr-1+ Cells in Mice With Myocardial Infarction.

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Hydrogen Sulfide Reduces Recru ...... ce With Myocardial Infarction.

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Hydrogen Sulfide Reduces Recru ...... ce With Myocardial Infarction.

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Hydrogen Sulfide Reduces Recru ...... ce With Myocardial Infarction.

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Hydrogen Sulfide Reduces Recru ...... ce With Myocardial Infarction.

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Hydrogen Sulfide Reduces Recru ...... ce With Myocardial Infarction.

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Hydrogen Sulfide Reduces Recru ...... ce With Myocardial Infarction.

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Hydrogen Sulfide Reduces Recru ...... ce With Myocardial Infarction.

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P2093

Bing Wu

http://www.w3.org/2001/XMLSchema#string

Hua Li

http://www.w3.org/2001/XMLSchema#string

Jia-Ning Wang

http://www.w3.org/2001/XMLSchema#string

Lei Zhang

http://www.w3.org/2001/XMLSchema#string

San-Wu Wu

http://www.w3.org/2001/XMLSchema#string

Ting Wu

http://www.w3.org/2001/XMLSchema#string

You-En Zhang

http://www.w3.org/2001/XMLSchema#string

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Myeloid-derived suppressor cells as regulators of the immune system

Anti-apoptotic and anti-inflammatory effects of hydrogen sulfide in a rat model of regional myocardial I/R

The healing myocardium sequentially mobilizes two monocyte subsets with divergent and complementary functions

Genetic and pharmacologic hydrogen sulfide therapy attenuates ischemia-induced heart failure in mice.

H2S ameliorates oxidative and proteolytic stresses and protects the heart against adverse remodeling in chronic heart failure

CCR5 signaling suppresses inflammation and reduces adverse remodeling of the infarcted heart, mediating recruitment of regulatory T cells

Antigen-specific inhibition of CD8+ T cell response by immature myeloid cells in cancer is mediated by reactive oxygen species.

Role of the complement system in ischaemic heart disease: potential for pharmacological intervention.

The effects of therapeutic sulfide on myocardial apoptosis in response to ischemia-reperfusion injury

H₂S protects against pressure overload-induced heart failure via upregulation of endothelial nitric oxide synthase.

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Hydrogen Sulfide Reduces Recruitment of CD11b+Gr-1+ Cells in Mice With Myocardial Infarction.

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P2860

P2860

Hydrogen Sulfide Reduces Recruitment of CD11b+Gr-1+ Cells in Mice With Myocardial Infarction.

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