about
Serum levels of leptin and high molecular weight adiponectin are inversely associated with radiographic spinal progression in patients with ankylosing spondylitis: results from the ENRADAS trialRegulation and migratory role of P-selectin ligands during intestinal inflammation.In vivo pre-activation of monocytes in patients with axial spondyloarthritisCartilage in facet joints of patients with ankylosing spondylitis (AS) shows signs of cartilage degeneration rather than chondrocyte hypertrophy: implications for joint remodeling in ASTNF signaling drives myeloid-derived suppressor cell accumulation.Enhanced antitumor immunity in mice deficient in CD69.Anti-RANKL treatment inhibits erosive joint destruction and lowers inflammation but has no effect on bone formation in the delayed-type hypersensitivity arthritis (DTHA) modelCore 2 ß1,6-N-acetylglucosaminyltransferase-I, crucial for P-selectin ligand expression is controlled by a distal enhancer regulated by STAT4 and T-bet in CD4+ T helper cells 1.Identification of two regulatory elements controlling Fucosyltransferase 7 transcription in murine CD4+ T cells.Bone marrow Th17 TNFα cells induce osteoclast differentiation and link bone destruction to IBD.Cellular players and role of selectin ligands in leukocyte recruitment in a T-cell-initiated delayed-type hypersensitivity reaction.Migration matters: regulatory T-cell compartmentalization determines suppressive activity in vivo.Differential regulation of P-selectin ligand expression in naive versus memory CD4+ T cells: evidence for epigenetic regulation of involved glycosyltransferase genes.Expression of selectin ligands on murine effector and IL-10-producing CD4+ T cells from non-infected and infected tissues.In situ analysis of interleukin-23- and interleukin-12-positive cells in the spine of patients with ankylosing spondylitis.
P50
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