Laquinimod ameliorates excitotoxic damage by regulating glutamate re-uptake.

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Laquinimod ameliorates excitotoxic damage by regulating glutamate re-uptake.

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2018 nî lūn-bûn

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2018年の論文

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2018年学术文章

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2018年学术文章

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2018年学术文章

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2018年学术文章

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2018年学术文章

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2018年学术文章

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2018年學術文章

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2018年學術文章

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Laquinimod ameliorates excitotoxic damage by regulating glutamate re-uptake.

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Laquinimod ameliorates excitotoxic damage by regulating glutamate re-uptake.

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Laquinimod ameliorates excitotoxic damage by regulating glutamate re-uptake.

@en

Laquinimod ameliorates excitotoxic damage by regulating glutamate re-uptake.

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Laquinimod ameliorates excitotoxic damage by regulating glutamate re-uptake.

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Laquinimod ameliorates excitotoxic damage by regulating glutamate re-uptake.

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P1476

Laquinimod ameliorates excitotoxic damage by regulating glutamate re-uptake.

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P2093

Alessandra Musella

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Georgia Mandolesi

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Silvia Bullitta

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The concentrations and distributions of three C-terminal variants of the GLT1 (EAAT2; slc1a2) glutamate transporter protein in rat brain tissue suggest differential regulation

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Yin Yang 1 is a repressor of glutamate transporter EAAT2, and it mediates manganese-induced decrease of EAAT2 expression in astrocytes

Neuroinflammation and M2 microglia: the good, the bad, and the inflamed.

In vivo evidence of glutamate toxicity in multiple sclerosis.

Placebo-controlled trial of oral laquinimod for multiple sclerosis.

P2888

s12974-017-1048-6

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scholarly article

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10.1186/S12974-017-1048-6

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P433

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P478

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Francesca Romana Rizzo

P577

2018-01-05T00:00:00Z

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P6179

1100202149

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29304807

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P932

5756343

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