Host Tumor Suppressor p18INK4c functions as a potent cell-intrinsic inhibitor of γHV68 reactivation and pathogenesis.

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Host Tumor Suppressor p18INK4c functions as a potent cell-intrinsic inhibitor of γHV68 reactivation and pathogenesis.

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2018 nî lūn-bûn

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2018年の論文

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2018年学术文章

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2018年学术文章

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2018年学术文章

@zh-cn

2018年学术文章

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2018年学术文章

@zh-my

2018年学术文章

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2018年學術文章

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2018年學術文章

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name

Host Tumor Suppressor p18INK4c ...... reactivation and pathogenesis.

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Host Tumor Suppressor p18INK4c ...... reactivation and pathogenesis.

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Item

label

Host Tumor Suppressor p18INK4c ...... reactivation and pathogenesis.

@en

Host Tumor Suppressor p18INK4c ...... reactivation and pathogenesis.

@nl

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Host Tumor Suppressor p18 Func ...... Reactivation and Pathogenesis

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Host Tumor Suppressor p18INK4c ...... reactivation and pathogenesis.

@en

Host Tumor Suppressor p18INK4c ...... reactivation and pathogenesis.

@nl

P1476

Host Tumor Suppressor p18INK4c ...... reactivation and pathogenesis.

@en

P2093

Carlyne D Cool

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Darby G Oldenburg

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Eric T Clambey

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Eva M Medina

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Lauren M Oko

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P2860

Structural basis of inhibition of CDK-cyclin complexes by INK4 inhibitors

Novel INK4 proteins, p19 and p18, are specific inhibitors of the cyclin D-dependent kinases CDK4 and CDK6

Complete sequence and genomic analysis of murine gammaherpesvirus 68

Silencing of the p18INK4c gene by promoter hypermethylation in Reed-Sternberg cells in Hodgkin lymphomas

Cdks, cyclins and CKIs: roles beyond cell cycle regulation

Viral cyclins mediate separate phases of infection by integrating functions of distinct mammalian cyclins

Virus reconstituted from infectious bacterial artificial chromosome (BAC)-cloned murine gammaherpesvirus 68 acquires wild-type properties in vivo only after excision of BAC vector sequences.

Epstein-Barr virus (EBV) reactivation is a frequent event after allogeneic stem cell transplantation (SCT) and quantitatively predicts EBV-lymphoproliferative disease following T-cell--depleted SCT.

Virus-encoded microRNAs facilitate gammaherpesvirus latency and pathogenesis in vivo

Involvement of TLR2 in recognition of acute gammaherpesvirus-68 infection.

P31

scholarly article

P356

10.1128/JVI.01604-17

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P407

English

P433

http://www.w3.org/2001/XMLSchema#string

P478

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P50

Douglas W White

Brian F Niemeyer

Linda F van Dyk

P577

2018-01-03T00:00:00Z

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P698

29298882

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P921

immunology

virology

P932

5827403

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