Deletion of glutamate dehydrogenase 1 (Glud1) in the central nervous system affects glutamate handling without altering synaptic transmission.
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De novo Synthesis of Glial Glutamate and Glutamine in Young Mice Requires Aspartate Provided by the Neuronal Mitochondrial Aspartate-Glutamate Carrier Aralar/AGC1Interaction of glutaric aciduria type 1-related glutaryl-CoA dehydrogenase with mitochondrial matrix proteinsThe Glutamate-Glutamine (GABA) Cycle: Importance of Late Postnatal Development and Potential Reciprocal Interactions between Biosynthesis and Degradation.Glutamine-Glutamate Cycle Flux Is Similar in Cultured Astrocytes and Brain and Both Glutamate Production and Oxidation Are Mainly Catalyzed by Aspartate AminotransferaseDetermination of glutamate dehydrogenase activity and its kinetics in mouse tissues using metabolic mapping (quantitative enzyme histochemistry)Regulation of brain glutamate metabolism by nitric oxide and S-nitrosylationAstrocytic-Neuronal-Astrocytic Pathway Selection for Formation and Degradation of Glutamate/GABAThe Glutamate Dehydrogenase Pathway and Its Roles in Cell and Tissue Biology in Health and Disease.Development of mice with brain-specific deletion of floxed glud1 (glutamate dehydrogenase 1) using cre recombinase driven by the nestin promoter.Astrocytic Control of Biosynthesis and Turnover of the Neurotransmitters Glutamate and GABA.Heterogeneous cellular distribution of glutamate dehydrogenase in brain and in non-neural tissues.Glutamate and ATP at the Interface Between Signaling and Metabolism in Astroglia: Examples from Pathology.Metabolic and Inflammatory Adaptation of Reactive Astrocytes: Role of PPARs.Glutamate oxidation in astrocytes: Roles of glutamate dehydrogenase and aminotransferases.2-Methylcitric acid impairs glutamate metabolism and induces permeability transition in brain mitochondria.Inhibitors of glutamate dehydrogenase block sodium-dependent glutamate uptake in rat brain membranes.α-Ketoadipic Acid and α-Aminoadipic Acid Cause Disturbance of Glutamatergic Neurotransmission and Induction of Oxidative Stress In Vitro in Brain of Adolescent Rats.Success: evolutionary and structural properties of amino acids prove effective for succinylation site prediction.Glutamate dehydrogenase is essential to sustain neuronal oxidative energy metabolism during stimulation.Glucose replaces glutamate as energy substrate to fuel glutamate uptake in glutamate dehydrogenase-deficient astrocytes.Glucose, Lactate, β-Hydroxybutyrate, Acetate, GABA, and Succinate as Substrates for Synthesis of Glutamate and GABA in the Glutamine-Glutamate/GABA Cycle.Widening Spectrum of Cellular and Subcellular Expression of Human GLUD1 and GLUD2 Glutamate Dehydrogenases Suggests Novel Functions.Dysfunctional TCA-Cycle Metabolism in Glutamate Dehydrogenase Deficient Astrocytes
P2860
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P2860
Deletion of glutamate dehydrogenase 1 (Glud1) in the central nervous system affects glutamate handling without altering synaptic transmission.
description
2012 nî lūn-bûn
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2012年の論文
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2012年学术文章
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2012年学术文章
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2012年学术文章
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2012年學術文章
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name
Deletion of glutamate dehydrog ...... ltering synaptic transmission.
@en
Deletion of glutamate dehydrogenase 1
@nl
type
label
Deletion of glutamate dehydrog ...... ltering synaptic transmission.
@en
Deletion of glutamate dehydrogenase 1
@nl
prefLabel
Deletion of glutamate dehydrog ...... ltering synaptic transmission.
@en
Deletion of glutamate dehydrogenase 1
@nl
P2093
P2860
P1476
Deletion of glutamate dehydrog ...... ltering synaptic transmission.
@en
P2093
Dominique Muller
Dorte M Skytt
Francesca Frigerio
Kamilla Pajęcka
Mathias De Roo
Melis Karaca
Pierre Maechler
Stefania Carobbio
Vladimír Mlynárik
P2860
P304
P356
10.1111/J.1471-4159.2012.07933.X
P407
P577
2012-09-28T00:00:00Z