B cell receptor signal strength determines B cell fate. - Wikidata - awgldk - TriplyDB

B cell receptor signal strength determines B cell fate.

B cell receptor signal strength determines B cell fate.

http://www.wikidata.org/entity/Q47221910

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Secretory IgA: Designed for Anti-Microbial Defense Accumulation of marginal zone B cells and accelerated loss of follicular dendritic cells in NF-kappaB p50-deficient mice.The transmembrane activator TACI triggers immunoglobulin class switching by activating B cells through the adaptor MyD88 Signalling of the BCR is regulated by a lipid rafts-localised transcription factor, Bright.Impaired B cell development and function in the absence of IkappaBNS Regulation of aicda expression and AID activity: relevance to somatic hypermutation and class switch DNA recombination Targeting B-cell receptor signaling kinases in chronic lymphocytic leukemia: the promise of entospletinib Translating transitions - how to decipher peripheral human B cell development S1PR2 links germinal center confinement and growth regulation Epstein-Barr virus and the origin of Hodgkin lymphoma Primary immunoglobulin repertoire development: time and space matter Epstein-Barr virus LMP2A reduces hyperactivation induced by LMP1 to restore normal B cell phenotype in transgenic mice Hepatitis C Virus (HCV)-Induced Immunoglobulin Hypermutation Reduces the Affinity and Neutralizing Activities of Antibodies against HCV Envelope Protein Regulation of frontline antibody responses by innate immune signals.FCRL5 exerts binary and compartment-specific influence on innate-like B-cell receptor signaling c-Abl-deficient mice exhibit reduced numbers of peritoneal B-1 cells and defects in BCR-induced B cell activation Mzb1 protein regulates calcium homeostasis, antibody secretion, and integrin activation in innate-like B cells M17, a gene specific for germinal center (GC) B cells and a prognostic marker for GC B-cell lymphomas, is dispensable for the GC reaction in mice Epstein-Barr virus LMP2A signaling in statu nascendi mimics a B cell antigen receptor-like activation signal Inflammatory monocytes hinder antiviral B cell responses.EBV latent membrane protein 1 activates Akt, NFkappaB, and Stat3 in B cell lymphomas.The SCHOOL of nature: I. Transmembrane signaling.Crucial role for BAFF-BAFF-R signaling in the survival and maintenance of mature B cells.Germinal center reutilization by newly activated B cells Progenitor cell origin plays a role in fate choices of mature B cells Development of Sjogren's syndrome in nonobese diabetic-derived autoimmune-prone C57BL/6.NOD-Aec1Aec2 mice is dependent on complement component-3.Epstein-Barr virus in Burkitt's lymphoma: a role for latent membrane protein 2A Pathogenetic importance and therapeutic implications of NF-κB in lymphoid malignancies.B cell TLRs and induction of immunoglobulin class-switch DNA recombination.Immunoglobulin responses at the mucosal interface.B cells and transplantation: an educational resource.Peripheral B cells latently infected with Epstein-Barr virus display molecular hallmarks of classical antigen-selected memory B cells.Epstein-Barr-virus-encoded LMP2A induces primary epithelial cell migration and invasion: possible role in nasopharyngeal carcinoma metastasis.Constitutive and B-cell receptor-induced activation of STAT3 are important signaling pathways targeted by bortezomib in leukemic mantle cell lymphoma Requirement for cyclin D3 in germinal center formation and function.Multiple factors influence the contribution of individual immunoglobulin light chain genes to the naïve antibody repertoire Responsiveness of B cells is regulated by the hinge region of IgD.Molecular biology of Hodgkin's and Reed/Sternberg cells in Hodgkin's lymphoma.Vitamin A-dependent transcriptional activation of the nuclear factor of activated T cells c1 (NFATc1) is critical for the development and survival of B1 cells The endocytic adaptor Eps15 controls marginal zone B cell numbers

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B cell receptor signal strength determines B cell fate.

http://www.wikidata.org/entity/Q47221910

description

2004 nî lūn-bûn

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2004年の論文

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2004年学术文章

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2004年学术文章

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2004年学术文章

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2004年學術文章

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2004年學術文章

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name

B cell receptor signal strength determines B cell fate.

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B cell receptor signal strength determines B cell fate.

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type

label

B cell receptor signal strength determines B cell fate.

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B cell receptor signal strength determines B cell fate.

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prefLabel

B cell receptor signal strength determines B cell fate.

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B cell receptor signal strength determines B cell fate.

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Nature Immunology

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B cell receptor signal strength determines B cell fate.

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Jeffery L Kutok

http://www.w3.org/2001/XMLSchema#string

Kevin L Otipoby

http://www.w3.org/2001/XMLSchema#string

Marat Alimzhanov

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Michael C Carroll

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Nathalie Uyttersprot

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Sibille Humme

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Stefano Casola

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P2860

A Toll-like receptor recognizes bacterial DNA

Interference with immunoglobulin (Ig)alpha immunoreceptor tyrosine-based activation motif (ITAM) phosphorylation modulates or blocks B cell development, depending on the availability of an Igbeta cytoplasmic tail

Epstein-Barr virus latent membrane protein 2A (LMP2A) employs the SLP-65 signaling module

Marginal-zone B cells

Origins and functions of B-1 cells with notes on the role of CD5

Complement. First of two parts

Receptor editing in a transgenic mouse model: site, efficiency, and role in B cell tolerance and antibody diversification

Class switch recombination and hypermutation require activation-induced cytidine deaminase (AID), a potential RNA editing enzyme

A cre-transgenic mouse strain for the ubiquitous deletion of loxP-flanked gene segments including deletion in germ cells

Germinal centers

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317-327

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14758357

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