Nlrp12 Mediates Adverse Neutrophil Recruitment during Influenza Virus Infection.

about

Nlrp12 Mediates Adverse Neutrophil Recruitment during Influenza Virus Infection.

Item

http://www.wikidata.org/entity/Q47241356

description

2017 nî lūn-bûn

@nan

2017年の論文

@ja

2017年学术文章

@wuu

2017年学术文章

@zh

2017年学术文章

@zh-cn

2017年学术文章

@zh-hans

2017年学术文章

@zh-my

2017年学术文章

@zh-sg

2017年學術文章

@yue

2017年學術文章

@zh-hant

name

Nlrp12 Mediates Adverse Neutrophil Recruitment during Influenza Virus Infection.

@en

Nlrp12 Mediates Adverse Neutrophil Recruitment during Influenza Virus Infection.

@nl

type

Item

label

Nlrp12 Mediates Adverse Neutrophil Recruitment during Influenza Virus Infection.

@en

Nlrp12 Mediates Adverse Neutrophil Recruitment during Influenza Virus Infection.

@nl

prefLabel

Nlrp12 Mediates Adverse Neutrophil Recruitment during Influenza Virus Infection.

@en

Nlrp12 Mediates Adverse Neutrophil Recruitment during Influenza Virus Infection.

@nl

P1476

Nlrp12 Mediates Adverse Neutrophil Recruitment during Influenza Virus Infection

@en

P2093

Ann M Miller

http://www.w3.org/2001/XMLSchema#string

Emma E Hornick

http://www.w3.org/2001/XMLSchema#string

Fayyaz S Sutterwala

http://www.w3.org/2001/XMLSchema#string

Kevin L Legge

http://www.w3.org/2001/XMLSchema#string

Mary E Wilson

http://www.w3.org/2001/XMLSchema#string

Suzanne L Cassel

http://www.w3.org/2001/XMLSchema#string

P2860

PYPAF7, a novel PYRIN-containing Apaf1-like protein that regulates activation of NF-kappa B and caspase-1-dependent cytokine processing

Cutting edge: Monarch-1: a pyrin/nucleotide-binding domain/leucine-rich repeat protein that controls classical and nonclassical MHC class I genes

Modulating the Innate Immune Response to Influenza A Virus: Potential Therapeutic Use of Anti-Inflammatory Drugs

Cell type- and stimulus-specific mechanisms for post-transcriptional control of neutrophil chemokine gene expression

Modeling host genetic regulation of influenza pathogenesis in the collaborative cross

NLRP12 suppresses colon inflammation and tumorigenesis through the negative regulation of noncanonical NF-κB signaling

Cutting edge: NLRP12 controls dendritic and myeloid cell migration to affect contact hypersensitivity

The NLRP12 Sensor Negatively Regulates Autoinflammatory Disease by Modulating Interleukin-4 Production in T Cells.

An absence of reactive oxygen species improves the resolution of lung influenza infection.

H5N1 and 1918 pandemic influenza virus infection results in early and excessive infiltration of macrophages and neutrophils in the lungs of mice.

P304

1188-1197

http://www.w3.org/2001/XMLSchema#string

P31

scholarly article

P356

10.4049/JIMMUNOL.1700999

http://www.w3.org/2001/XMLSchema#string

P407

English

P433

http://www.w3.org/2001/XMLSchema#string

P478

200

http://www.w3.org/2001/XMLSchema#string

P50

Balaji Banoth

Nidhi Jain

Gail A Bishop

Katherine N Gibson-Corley

Zeb R Zacharias

P577

2017-12-27T00:00:00Z

http://www.w3.org/2001/XMLSchema#dateTime

P698

29282312

http://www.w3.org/2001/XMLSchema#string

P932

5831365

http://www.w3.org/2001/XMLSchema#string