Oncogenic src, raf, and ras stimulate a hypertrophic pattern of gene expression and increase cell size in neonatal rat ventricular myocytes.
about
Focal adhesion kinase and p130Cas mediate both sarcomeric organization and activation of genes associated with cardiac myocyte hypertrophyAltered focal adhesion regulation correlates with cardiomyopathy in mice expressing constitutively active rac1Dominant negative Ras attenuates pathological ventricular remodeling in pressure overload cardiac hypertrophyRegulation of mitogen-activated protein kinases in cardiac myocytes through the small G protein Rac1c-Jun N-terminal kinase-interacting protein 1 inhibits gene expression in response to hypertrophic agonists in neonatal rat ventricular myocytesPhosphorylation of GATA-4 is involved in alpha 1-adrenergic agonist-responsive transcription of the endothelin-1 gene in cardiac myocytesNetwork reconstruction and systems analysis of cardiac myocyte hypertrophy signaling.Cardiac-specific overexpression of AT1 receptor mutant lacking G alpha q/G alpha i coupling causes hypertrophy and bradycardia in transgenic mice.Mechanical strain activates BNP gene transcription through a p38/NF-kappaB-dependent mechanism.A novel pleiotropic effect of statins: prevention of cardiac hypertrophy by cholesterol-independent mechanismsStatins and myocardial hypertrophy.Statin therapy for cardiac hypertrophy and heart failureStatins and the myocardiumStatins in non-ischaemic cardiomyopathy: an update on our current clinical and pathophysiological understanding.Functional thyrotropin receptor expression in the ventricle and the effects on ventricular BNP secretion.Agonist-dependent phosphorylation of the G protein-coupled receptor kinase 2 (GRK2) by Src tyrosine kinase.Stimulation of gene expression in neonatal rat ventricular myocytes by Ras is mediated by Ral guanine nucleotide dissociation stimulator (Ral.GDS) and phosphatidylinositol 3-kinase in addition to Raf.Aldosterone stimulates proliferation of cardiac fibroblasts by activating Ki-RasA and MAPK1/2 signaling.Endothelin-1 induces expression of fetal genes through the interleukin-6 family of cytokines in cardiac myocytes.Regulation of Ras·GTP Loading and Ras-Raf Association in Neonatal Rat Ventricular Myocytes by G Protein-coupled Receptor Agonists and Phorbol Ester
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P2860
Oncogenic src, raf, and ras stimulate a hypertrophic pattern of gene expression and increase cell size in neonatal rat ventricular myocytes.
description
1998 nî lūn-bûn
@nan
1998年の論文
@ja
1998年学术文章
@wuu
1998年学术文章
@zh
1998年学术文章
@zh-cn
1998年学术文章
@zh-hans
1998年学术文章
@zh-my
1998年学术文章
@zh-sg
1998年學術文章
@yue
1998年學術文章
@zh-hant
name
Oncogenic src, raf, and ras st ...... atal rat ventricular myocytes.
@en
Oncogenic src, raf, and ras st ...... atal rat ventricular myocytes.
@nl
type
label
Oncogenic src, raf, and ras st ...... atal rat ventricular myocytes.
@en
Oncogenic src, raf, and ras st ...... atal rat ventricular myocytes.
@nl
prefLabel
Oncogenic src, raf, and ras st ...... atal rat ventricular myocytes.
@en
Oncogenic src, raf, and ras st ...... atal rat ventricular myocytes.
@nl
P2093
P2860
P356
P1476
Oncogenic src, raf, and ras st ...... atal rat ventricular myocytes.
@en
P2093
Gillespie-Brown J
P2860
P304
18146-18152
P356
10.1074/JBC.273.29.18146
P407
P577
1998-07-01T00:00:00Z