BCL-XL binds and antagonizes RASSF6 tumor suppressor to suppress p53 expression.

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BCL-XL binds and antagonizes RASSF6 tumor suppressor to suppress p53 expression.

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2017 nî lūn-bûn

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2017年の論文

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2017年学术文章

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2017年学术文章

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2017年学术文章

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2017年学术文章

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2017年学术文章

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2017年学术文章

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2017年學術文章

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2017年學術文章

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BCL-XL binds and antagonizes RASSF6 tumor suppressor to suppress p53 expression.

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BCL-XL binds and antagonizes RASSF6 tumor suppressor to suppress p53 expression.

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BCL-XL binds and antagonizes RASSF6 tumor suppressor to suppress p53 expression.

@en

BCL-XL binds and antagonizes RASSF6 tumor suppressor to suppress p53 expression.

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BCL-XL binds and antagonizes RASSF6 tumor suppressor to suppress p53 expression.

@en

BCL-XL binds and antagonizes RASSF6 tumor suppressor to suppress p53 expression.

@nl

P1476

BCL-XL binds and antagonizes RASSF6 tumor suppressor to suppress p53 expression

@en

P2093

Aradhan Sarkar

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Hiroaki Iwasa

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Kyoko Arimoto-Matsuzaki

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Shakhawoat Hossain

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Xiaoyin Xu

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P2860

Bcl-2 family proteins regulate the release of apoptogenic cytochrome c by the mitochondrial channel VDAC

PUMA induces the rapid apoptosis of colorectal cancer cells

PUMA, a novel proapoptotic gene, is induced by p53

p53 has a direct apoptogenic role at the mitochondria

The tumor suppressor RASSF1A and MAP-1 link death receptor signaling to Bax conformational change and cell death

The conserved N-terminal BH4 domain of Bcl-2 homologues is essential for inhibition of apoptosis and interaction with CED-4

Bad, a heterodimeric partner for Bcl-XL and Bcl-2, displaces Bax and promotes cell death

bcl-x, a bcl-2-related gene that functions as a dominant regulator of apoptotic cell death

Bcl-2 heterodimerizes in vivo with a conserved homolog, Bax, that accelerates programmed cell death

BID: a novel BH3 domain-only death agonist

P304

993-1003

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P31

scholarly article

P356

10.1111/GTC.12541

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P433

http://www.w3.org/2001/XMLSchema#string

P478

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P50

Junichi Maruyama

Yutaka Hata

P577

2017-11-28T00:00:00Z

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P698

29193479

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