Neurons derived from sporadic Alzheimer's disease iPSCs reveal elevated TAU hyperphosphorylation, increased amyloid levels, and GSK3B activation.

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The Amyloid-β Oligomer Hypothesis: Beginning of the Third Decade.Regenerative potential of induced pluripotent stem cells derived from patients undergoing haemodialysis in kidney regeneration iPSC Modeling of Presenilin1 Mutation in Alzheimer's Disease with Cerebellar Ataxia

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Neurons derived from sporadic Alzheimer's disease iPSCs reveal elevated TAU hyperphosphorylation, increased amyloid levels, and GSK3B activation.

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2017 nî lūn-bûn

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2017年の論文

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2017年学术文章

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2017年学术文章

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2017年学术文章

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2017年学术文章

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2017年学术文章

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name

Neurons derived from sporadic ...... levels, and GSK3B activation.

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Neurons derived from sporadic ...... levels, and GSK3B activation.

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type

Item

label

Neurons derived from sporadic ...... levels, and GSK3B activation.

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Neurons derived from sporadic ...... levels, and GSK3B activation.

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prefLabel

Neurons derived from sporadic ...... levels, and GSK3B activation.

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Neurons derived from sporadic ...... levels, and GSK3B activation.

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P1476

Neurons derived from sporadic ...... d levels, and GSK3B activation

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P2093

Anna Ochalek

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Annamária Téglási

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Balázs Mihalik

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Bjørn Holst

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Hasan X Avci

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Jørgen E Nielsen

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Maria Lo Giudice

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Poul Hyttel

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Zsuzsanna Táncos

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P2860

Primed phosphorylation of tau at Thr231 by glycogen synthase kinase 3beta (GSK3beta) plays a critical role in regulating tau's ability to bind and stabilize microtubules

MARK, a novel family of protein kinases that phosphorylate microtubule-associated proteins and trigger microtubule disruption

Protein kinase MARK/PAR-1 is required for neurite outgrowth and establishment of neuronal polarity.

The structure and function of proline-rich regions in proteins

beta-Amyloid-(1-42) is a major component of cerebrovascular amyloid deposits: implications for the pathology of Alzheimer disease

Fast and accurate short read alignment with Burrows-Wheeler transform

Loss-of-function presenilin mutations in Alzheimer disease. Talking Point on the role of presenilin mutations in Alzheimer disease

Modeling Alzheimer's Disease with Induced Pluripotent Stem Cells: Current Challenges and Future Concerns

The amyloid hypothesis of Alzheimer's disease at 25 years

Crystal structure of glycogen synthase kinase 3 beta: structural basis for phosphate-primed substrate specificity and autoinhibition

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s13195-017-0317-z

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scholarly article

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10.1186/S13195-017-0317-Z

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English

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Abinaya Chandrasekaran

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2017-12-01T00:00:00Z

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1092759415

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29191219

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Alzheimer's disease

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5709977

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Neurons derived from sporadic Alzheimer's disease iPSCs reveal elevated TAU hyperphosphorylation, increased amyloid levels, and GSK3B activation.

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P2860

P2860

Neurons derived from sporadic Alzheimer's disease iPSCs reveal elevated TAU hyperphosphorylation, increased amyloid levels, and GSK3B activation.

description

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