Mithramycin A Enhances Tumor Sensitivity to Mitotic Catastrophe Resulting From DNA Damage.

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Mithramycin A Enhances Tumor Sensitivity to Mitotic Catastrophe Resulting From DNA Damage.

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2017 nî lūn-bûn

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2017年の論文

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2017年学术文章

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Mithramycin A Enhances Tumor S ...... phe Resulting From DNA Damage.

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Mithramycin A Enhances Tumor S ...... phe Resulting From DNA Damage.

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Mithramycin A Enhances Tumor S ...... phe Resulting From DNA Damage.

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Mithramycin A Enhances Tumor S ...... phe Resulting From DNA Damage.

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Mithramycin A Enhances Tumor S ...... phe Resulting From DNA Damage.

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Mithramycin A Enhances Tumor S ...... phe Resulting From DNA Damage.

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P1476

Mithramycin A Enhances Tumor S ...... phe Resulting From DNA Damage.

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Ayla O White

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Bradley T Scroggins

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Darmood Wei

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Deborah E Citrin

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Eun Joo Chung

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Grace McKay-Corkum

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Jeffrey Burkeen

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Kathryn E Hudak

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Luca F Valle

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Shilpa S Patil

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P2860

Control of apoptosis and mitotic spindle checkpoint by survivin

Survivin is required for a sustained spindle checkpoint arrest in response to lack of tension

MITHRAMYCIN IN THE TREATMENT OF DISSEMINATED TESTICULAR NEOPLASMS

Mithramycin A sensitizes cancer cells to TRAIL-mediated apoptosis by down-regulation of XIAP gene promoter through Sp1 sites

Mithramycin Depletes Specificity Protein 1 and Activates p53 to Mediate Senescence and Apoptosis of Malignant Pleural Mesothelioma Cells

Genome-wide modulation of gene transcription in ovarian carcinoma cells by a new mithramycin analogue

Radiation-induced cell death mechanisms.

Mithramycin inhibits SP1 binding and selectively inhibits transcriptional activity of the dihydrofolate reductase gene in vitro and in vivo.

Two molecularly distinct G(2)/M checkpoints are induced by ionizing irradiation

Sp1 facilitates DNA double-strand break repair through a nontranscriptional mechanism

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scholarly article

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10.1016/J.IJROBP.2017.09.049

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English

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2017-10-12T00:00:00Z

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29157749

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5777897

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