Pioglitazone limits myocardial infarct size, activates Akt, and upregulates cPLA2 and COX-2 in a PPAR-γ-independent manner.
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Peroxisome Proliferator-Activated Receptor-γ Is Critical to Cardiac FibrosisPPARs: Protectors or Opponents of Myocardial Function?Effects of PPARs agonists on cardiac metabolism in littermate and cardiomyocyte-specific PPAR-γ-knockout (CM-PGKO) mice.Acute administration of n-3 rich triglyceride emulsions provides cardioprotection in murine models after ischemia-reperfusionDisruption of group IVA cytosolic phospholipase A(2) attenuates myocardial ischemia-reperfusion injury partly through inhibition of TNF-α-mediated pathway.Metabolic stress in the myocardium: adaptations of gene expression.Drug Signature-based Finding of Additional Clinical Use of LC28-0126 for Neutrophilic Bronchial Asthma.Thiazolidinediones are acute, specific inhibitors of the mitochondrial pyruvate carrier.Cardiac vulnerability to ischemia/reperfusion injury drastically increases in late pregnancy.Pioglitazone reduces inflammation through inhibition of NF-κB in polymicrobial sepsis.Aleglitazar, a dual peroxisome proliferator-activated receptor-α and -γ agonist, protects cardiomyocytes against the adverse effects of hyperglycaemia.The potential effects of anti-diabetic medications on myocardial ischemia-reperfusion injury.Regulation of pyruvate metabolism in metabolic-related diseases.Knockout mouse models of insulin signaling: Relevance past and futurePharmacologic therapy for reducing myocardial infarct size in clinical trials: failed and promising approaches.The Role of Peroxisome Proliferator-Activated Receptor γ in Mediating Cardioprotection Against Ischemia/Reperfusion Injury.Effects of PPAR γ Agonist Pioglitazone on Redox-Sensitive Cellular Signaling in Young Spontaneously Hypertensive Rats.Pioglitazone attenuates progression of aortic valve calcification via down-regulating receptor for advanced glycation end products.Aleglitazar, a Balanced Dual PPARα and -γ Agonist, Protects the Heart Against Ischemia-Reperfusion Injury.Dickkopf-1 (DKK1) phosphatase and tensin homolog on chromosome 10 (PTEN) crosstalk via microRNA interference in the diabetic heart.Chronic Akt activation accentuates aging-induced cardiac hypertrophy and myocardial contractile dysfunction: role of autophagy.
P2860
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P2860
Pioglitazone limits myocardial infarct size, activates Akt, and upregulates cPLA2 and COX-2 in a PPAR-γ-independent manner.
description
2011 nî lūn-bûn
@nan
2011年の論文
@ja
2011年学术文章
@wuu
2011年学术文章
@zh
2011年学术文章
@zh-cn
2011年学术文章
@zh-hans
2011年学术文章
@zh-my
2011年学术文章
@zh-sg
2011年學術文章
@yue
2011年學術文章
@zh-hant
name
Pioglitazone limits myocardial ...... n a PPAR-γ-independent manner.
@en
Pioglitazone limits myocardial ...... n a PPAR-γ-independent manner.
@nl
type
label
Pioglitazone limits myocardial ...... n a PPAR-γ-independent manner.
@en
Pioglitazone limits myocardial ...... n a PPAR-γ-independent manner.
@nl
prefLabel
Pioglitazone limits myocardial ...... n a PPAR-γ-independent manner.
@en
Pioglitazone limits myocardial ...... n a PPAR-γ-independent manner.
@nl
P2093
P2860
P1476
Pioglitazone limits myocardial ...... n a PPAR-γ-independent manner.
@en
P2093
Jinqiao Qian
Jose R Perez-Polo
Yochai Birnbaum
P2860
P2888
P304
P356
10.1007/S00395-011-0162-3
P577
2011-03-01T00:00:00Z