Myocardial infarct size-limiting effect of chronic hypoxia persists for five weeks of normoxic recovery.
about
Mitochondrial BKCa channels contribute to protection of cardiomyocytes isolated from chronically hypoxic rats.Therapeutic potential of intermittent hypoxia: a matter of dose.Extending injury- and disease-resistant CNS phenotypes by repetitive epigenetic conditioning.ZFP580, a novel zinc-finger transcription factor, is involved in cardioprotection of intermittent high-altitude hypoxia against myocardial ischemia-reperfusion injury.Hypoxic Conditioning as a New Therapeutic Modality.Intermittent hypoxia conditioning protects mitochondrial cytochrome c oxidase of rat cerebellum from ethanol withdrawal stress.Enhancement of Glucose Metabolism via PGC-1α Participates in the Cardioprotection of Chronic Intermittent Hypobaric Hypoxia.Pathogenesis of myocardial ischemia-reperfusion injury and rationale for therapy.Hypoxia impairs agonist-induced integrin αIIbβ3 activation and platelet aggregation.Recognized Obstructive Sleep Apnea is Associated With Improved In-Hospital Outcomes After ST Elevation Myocardial Infarction.Hypoxia training attenuates left ventricular remodeling in rabbit with myocardial infarction.Enhancing and Extending Biological Performance and Resilience
P2860
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P2860
Myocardial infarct size-limiting effect of chronic hypoxia persists for five weeks of normoxic recovery.
description
2004 nî lūn-bûn
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2004年の論文
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name
Myocardial infarct size-limiti ...... ve weeks of normoxic recovery.
@en
Myocardial infarct size-limiti ...... ve weeks of normoxic recovery.
@nl
type
label
Myocardial infarct size-limiti ...... ve weeks of normoxic recovery.
@en
Myocardial infarct size-limiti ...... ve weeks of normoxic recovery.
@nl
prefLabel
Myocardial infarct size-limiti ...... ve weeks of normoxic recovery.
@en
Myocardial infarct size-limiti ...... ve weeks of normoxic recovery.
@nl
P2093
P1476
Myocardial infarct size-limiti ...... ve weeks of normoxic recovery.
@en
P2093
P304
P577
2004-01-01T00:00:00Z