Stress hormones promote EGFR inhibitor resistance in NSCLC: Implications for combinations with β-blockers.
about
Mechanisms and clinical activity of an EGFR and HER2 exon 20-selective kinase inhibitor in non-small cell lung cancer.Cigarette smoke enhances oncogene addiction to c-MET and desensitizes EGFR-expressing non-small cell lung cancer to EGFR TKIs.Stressing the need to overcome EGFR tyrosine kinase inhibitor resistance.Repurposing established cyclic adenosine monophosphate reducing agents for the prevention and therapy of epidermal growth factor receptor inhibitor resistance in non-small cell lung cancer.Propranolol for the treatment of vascular sarcomas
P2860
Stress hormones promote EGFR inhibitor resistance in NSCLC: Implications for combinations with β-blockers.
description
2017 nî lūn-bûn
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2017年の論文
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name
Stress hormones promote EGFR i ...... combinations with β-blockers.
@en
Stress hormones promote EGFR i ...... combinations with β-blockers.
@nl
type
label
Stress hormones promote EGFR i ...... combinations with β-blockers.
@en
Stress hormones promote EGFR i ...... combinations with β-blockers.
@nl
prefLabel
Stress hormones promote EGFR i ...... combinations with β-blockers.
@en
Stress hormones promote EGFR i ...... combinations with β-blockers.
@nl
P2093
P2860
P50
P1476
Stress hormones promote EGFR i ...... combinations with β-blockers.
@en
P2093
Anil K Sood
Daniel Gomez
Guillermo Armaiz Pena
Kathryn Howells
Roy Herbst
Waun Ki Hong
Youhong Fan
P2860
P356
10.1126/SCITRANSLMED.AAO4307
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P50
P577
2017-11-01T00:00:00Z