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Chromatin organization in pluripotent cells: emerging approaches to study and disrupt functionEarly cell fate decisions of human embryonic stem cells and mouse epiblast stem cells are controlled by the same signalling pathways.Global chromatin architecture reflects pluripotency and lineage commitment in the early mouse embryo.Transcriptional response of Hoxb genes to retinoid signalling is regionally restricted along the neural tube rostrocaudal axis.Distinct histone modifications in stem cell lines and tissue lineages from the early mouse embryoCharacterization of human embryonic stem cell lines by the International Stem Cell Initiative.Comprehensive Cell Surface Protein Profiling Identifies Specific Markers of Human Naive and Primed Pluripotent StatesThe pluripotency factor Nanog regulates pericentromeric heterochromatin organization in mouse embryonic stem cellsAnnual meeting of the EpiGeneSys Network of Excellence--Advancing epigenetics towards systems biology.Comparative Principles of DNA Methylation Reprogramming during Human and Mouse In Vitro Primordial Germ Cell Specification.XACT Noncoding RNA Competes with XIST in the Control of X Chromosome Activity during Human Early Development.Gata3 regulates trophoblast development downstream of Tead4 and in parallel to Cdx2.Crosstalk between pluripotency factors and higher-order chromatin organization.DNA methylation is dispensable for changes in global chromatin architecture but required for chromocentre formation in early stem cell differentiation.Deletion of the Polycomb-Group Protein EZH2 Leads to Compromised Self-Renewal and Differentiation Defects in Human Embryonic Stem Cells.Assessing the Safety of Human Pluripotent Stem Cells and Their Derivatives for Clinical Applications.The challenge of regulating rapidly changing science: stem cell legislation in Canada.Recombination signatures distinguish embryonic stem cells derived by parthenogenesis and somatic cell nuclear transfer.Epigenetic status of human embryonic stem cells.Cell-surface proteomics identifies lineage-specific markers of embryo-derived stem cells.Global reorganisation of cis-regulatory units upon lineage commitment of human embryonic stem cells.Activin/Nodal signalling maintains pluripotency by controlling Nanog expression.Naive pluripotent stem cells as a model for studying human developmental epigenomics: opportunities and limitations.Derivation and Culture of Epiblast Stem Cell (EpiSC) Lines.Derivation and Culture of Extra-Embryonic Endoderm Stem Cell Lines.Molecular profiling of aged neural progenitors identifies Dbx2 as a candidate regulator of age-associated neurogenic decline.Identifying Human Naïve Pluripotent Stem Cells - Evaluating State-Specific Reporter Lines and Cell-Surface Markers.Long-Range Enhancer Interactions Are Prevalent in Mouse Embryonic Stem Cells and Are Reorganized upon Pluripotent State Transition.Promoter interactome of human embryonic stem cell-derived cardiomyocytes connects GWAS regions to cardiac gene networks.Author Correction: Promoter interactome of human embryonic stem cell-derived cardiomyocytes connects GWAS regions to cardiac gene networksTranscription factors make the right contactsGenome-Scale Oscillations in DNA Methylation during Exit from Pluripotency
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P50
description
hulumtues
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հետազոտող
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Peter J Rugg-Gunn
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Peter J Rugg-Gunn
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Peter J Rugg-Gunn
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Peter J Rugg-Gunn
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Peter J Rugg-Gunn
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Peter J Rugg-Gunn
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Peter J Rugg-Gunn
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Peter J Rugg-Gunn
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Peter J Rugg-Gunn
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Peter J Rugg-Gunn
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Peter J Rugg-Gunn
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Peter J Rugg-Gunn
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Peter J Rugg-Gunn
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Peter J Rugg-Gunn
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Peter J Rugg-Gunn
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Peter J Rugg-Gunn
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Peter J Rugg-Gunn
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Peter J Rugg-Gunn
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P106
P21
P31
P496
0000-0002-9601-5949