about
Genetically-determined hyperfunction of the S100B/RAGE axis is a risk factor for aspergillosis in stem cell transplant recipientsPaving the way for predictive diagnostics and personalized treatment of invasive aspergillosisIL-22 and IDO1 affect immunity and tolerance to murine and human vaginal candidiasisGenetic Variation in Autophagy-Related Genes Influences the Risk and Phenotype of Buruli UlcerDectin-1 isoforms contribute to distinct Th1/Th17 cell activation in mucosal candidiasis.Impact of Paracoccin Gene Silencing on Paracoccidioides brasiliensis Virulence.The C allele of rs5743836 polymorphism in the human TLR9 promoter links IL-6 and TLR9 up-regulation and confers increased B-cell proliferationTLR9 activation dampens the early inflammatory response to Paracoccidioides brasiliensis, impacting host survival.Human genetic susceptibility to invasive aspergillosis.Characterization of specific immune responses to different Aspergillus antigens during the course of invasive Aspergillosis in hematologic patientsNitric oxide signaling is disrupted in the yeast model for Batten disease.CD4(+) T cell vaccination overcomes defective cross-presentation of fungal antigens in a mouse model of chronic granulomatous disease.DAMP signaling in fungal infections and diseases.Polymorphisms in Host Immunity-Modulating Genes and Risk of Invasive Aspergillosis: Results from the AspBIOmics Consortium.Association of a variable number tandem repeat in the NLRP3 gene in women with susceptibility to RVVCThe soluble pattern recognition receptor PTX3 links humoral innate and adaptive immune responses by helping marginal zone B cells.Immunometabolic Pathways in BCG-Induced Trained Immunity.Genetic variability of innate immunity impacts human susceptibility to fungal diseases.Non-hematopoietic cells contribute to protective tolerance to Aspergillus fumigatus via a TRIF pathway converging on IDO.Cracking the Toll-like receptor code in fungal infections.Immunity and tolerance to infections in experimental hematopoietic transplantation.Immunogenetic profiling to predict risk of invasive fungal diseases: where are we now?From memory to antifungal vaccine design.Neutrophil responses to aspergillosis: new roles for old players.The microbiome-metabolome crosstalk in the pathogenesis of respiratory fungal diseases.Glutaminolysis and Fumarate Accumulation Integrate Immunometabolic and Epigenetic Programs in Trained Immunity.The Cell Biology of the Trichosporon-Host Interaction.Th17/Treg imbalance in murine cystic fibrosis is linked to indoleamine 2,3-dioxygenase deficiency but corrected by kynurenines.Towards a molecular genetic system for the pathogenic fungus Paracoccidioides brasiliensis.IL-10 overexpression predisposes to invasive aspergillosis by suppressing antifungal immunity.Host-Derived Biomarkers for Risk Assessment of Invasive Fungal Diseases.Immunity and tolerance to fungi in hematopoietic transplantation: principles and perspectives.Host defense pathways against fungi: the basis for vaccines and immunotherapy.Genetic PTX3 deficiency and aspergillosis in stem-cell transplantation.Correction: The soluble pattern recognition receptor PTX3 links humoral innate and adaptive immune responses by helping marginal zone B cells.Editorial: An Omics Perspective on Fungal Infection: Toward Next-Generation Diagnosis and Therapy.Intranasally delivered siRNA targeting PI3K/Akt/mTOR inflammatory pathways protects from aspergillosis.T-1237C polymorphism of TLR9 gene is not associated with multiple sclerosis in the Portuguese population.Inflammation in aspergillosis: the good, the bad, and the therapeutic.Hypoxia promotes danger-mediated inflammation via receptor for advanced glycation end products in cystic fibrosis.
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description
hulumtues
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onderzoeker
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հետազոտող
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Agostinho Carvalho
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Agostinho Carvalho
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Agostinho Carvalho
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Agostinho Carvalho
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Agostinho Carvalho
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Agostinho Carvalho
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Agostinho Carvalho
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Agostinho Carvalho
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Agostinho Carvalho
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Agostinho Carvalho
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Agostinho Carvalho
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Agostinho Carvalho
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Agostinho Carvalho
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Agostinho Carvalho
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Agostinho Carvalho
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