FM19G11 favors spinal cord injury regeneration and stem cell self-renewal by mitochondrial uncoupling and glucose metabolism induction.
about
A specialized microvascular domain in the mouse neural stem cell niche.FM19G11 reverses endothelial dysfunction in rat and human arteries through stimulation of the PI3K/Akt/eNOS pathway, independently of mTOR/HIF-1α activation.(18)F-FDG-PET imaging of rat spinal cord demonstrates altered glucose uptake acutely after contusion injury.Stem Cells and Labeling for Spinal Cord Injury.Fgf2 improves functional recovery-decreasing gliosis and increasing radial glia and neural progenitor cells after spinal cord injury.Cocktail of chemical compounds robustly promoting cell reprogramming protects liver against acute injuryThe multifaceted activities of AMPK in tumor progression--why the "one size fits all" definition does not fit at all?Prospects for therapeutic mitochondrial transplantation.Distinct Effects of miR-210 Reduction on Neurogenesis: Increased Neuronal Survival of Inflammation But Reduced Proliferation Associated with Mitochondrial Enhancement.Priming of the Cells: Hypoxic Preconditioning for Stem Cell TherapyNovel direct AMPK activator suppresses non-small cell lung cancer through inhibition of lipid metabolism.Optimization of mitochondrial isolation techniques for intraspinal transplantation procedures.Connexin 50 modulates Sox2 expression in spinal-cord-derived ependymal stem/progenitor cells.Methacrylate-endcapped caprolactone and FM19G11 provide a proper niche for spinal cord-derived neural cells.FM19G11 and Ependymal Progenitor/Stem Cell Combinatory Treatment Enhances Neuronal Preservation and Oligodendrogenesis after Severe Spinal Cord Injury.Effects of Mitochondrial Transplantation on Bioenergetics, Cellular Incorporation, and Functional Recovery after Spinal Cord Injury.Stem Cells Therapy for Spinal Cord Injury.
P2860
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P2860
FM19G11 favors spinal cord injury regeneration and stem cell self-renewal by mitochondrial uncoupling and glucose metabolism induction.
description
2012 nî lūn-bûn
@nan
2012年の論文
@ja
2012年学术文章
@wuu
2012年学术文章
@zh
2012年学术文章
@zh-cn
2012年学术文章
@zh-hans
2012年学术文章
@zh-my
2012年学术文章
@zh-sg
2012年學術文章
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2012年學術文章
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name
FM19G11 favors spinal cord inj ...... glucose metabolism induction.
@en
FM19G11 favors spinal cord inj ...... glucose metabolism induction.
@nl
type
label
FM19G11 favors spinal cord inj ...... glucose metabolism induction.
@en
FM19G11 favors spinal cord inj ...... glucose metabolism induction.
@nl
prefLabel
FM19G11 favors spinal cord inj ...... glucose metabolism induction.
@en
FM19G11 favors spinal cord inj ...... glucose metabolism induction.
@nl
P2093
P2860
P356
P1433
P1476
FM19G11 favors spinal cord inj ...... glucose metabolism induction.
@en
P2093
Ana Alastrue-Agudo
Francisco Javier Rodríguez-Jimnez
Miodrag Stojkovic
Slaven Erceg
Victoria Moreno-Manzano
P2860
P304
P356
10.1002/STEM.1189
P577
2012-10-01T00:00:00Z