Targeting glioma stem cells through combined BMI1 and EZH2 inhibition. - Wikidata - awgldk - TriplyDB

Targeting glioma stem cells through combined BMI1 and EZH2 inhibition.

Targeting glioma stem cells through combined BMI1 and EZH2 inhibition.

http://www.wikidata.org/entity/Q47648829

about

Targeting glioma stem-like cell survival and chemo-resistance through inhibition of lysine-specific histone demethylase KDM2B.Update on the role and therapeutic potential of polycomb repressive complexes in (biliary tract) cancer.Utility of Glioblastoma Patient-Derived Orthotopic Xenografts in Drug Discovery and Personalized Therapy.From Chemotherapy to Combined Targeted Therapeutics: In Vitro and in Vivo Models to Decipher Intra-tumor Heterogeneity.Apelin, the Devil Inside Brain Tumors.Re-thinking the preclinical development of GBM therapeutics.Stromal miR-20a controls paracrine CXCL8 secretion in colitis and colon cancer.Distinctive epigenomes characterize glioma stem cells and their response to differentiation cues.Transcriptional Repression of p53 by PAX3 Contributes to Gliomagenesis and Differentiation of Glioma Stem Cells.Aberrant neuronal differentiation is common in glioma but is associated neither with epileptic seizures nor with better survival Baicalin suppresses proliferation, migration, and invasion in human glioblastoma cells via Ca-dependent pathway Next-Generation Modeling of Human Cancers The Methylation Status of the Epigenome: Its Emerging Role in the Regulation of Tumor Angiogenesis and Tumor Growth, and Potential for Drug Targeting

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P2860

Targeting glioma stem cells through combined BMI1 and EZH2 inhibition.

http://www.wikidata.org/entity/Q47648829

description

2017 nî lūn-bûn

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2017年の論文

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2017年学术文章

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2017年学术文章

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2017年学术文章

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2017年学术文章

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2017年学术文章

@zh-my

2017年学术文章

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2017年學術文章

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2017年學術文章

@zh-hant

name

Targeting glioma stem cells through combined BMI1 and EZH2 inhibition.

@en

Targeting glioma stem cells through combined BMI1 and EZH2 inhibition.

@nl

type

label

Targeting glioma stem cells through combined BMI1 and EZH2 inhibition.

@en

Targeting glioma stem cells through combined BMI1 and EZH2 inhibition.

@nl

prefLabel

Targeting glioma stem cells through combined BMI1 and EZH2 inhibition.

@en

Targeting glioma stem cells through combined BMI1 and EZH2 inhibition.

@nl

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Nature Medicine

P1476

Targeting glioma stem cells through combined BMI1 and EZH2 inhibition.

@en

P2093

Andrew E Sloan

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Briana C Prager

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Claudia L Valentim

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Jeremy N Rich

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Leo J Y Kim

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Lisa C Wallace

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Ping Huang

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Qi-Gang Zhou

http://www.w3.org/2001/XMLSchema#string

Qiulian Wu

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Ryan C Gimple

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P2860

Phosphorylation of EZH2 activates STAT3 signaling via STAT3 methylation and promotes tumorigenicity of glioblastoma stem-like cells

Role of histone H2A ubiquitination in Polycomb silencing

Somatic histone H3 alterations in pediatric diffuse intrinsic pontine gliomas and non-brainstem glioblastomas

RYBP-PRC1 complexes mediate H2A ubiquitylation at polycomb target sites independently of PRC2 and H3K27me3

BMI-1 promotes ewing sarcoma tumorigenicity independent of CDKN2A repression

Integrated genomic analysis identifies clinically relevant subtypes of glioblastoma characterized by abnormalities in PDGFRA, IDH1, EGFR, and NF1

An inhibitor of mutant IDH1 delays growth and promotes differentiation of glioma cells.

Glioma stem cells promote radioresistance by preferential activation of the DNA damage response

Identification of human brain tumour initiating cells

RNF144A, an E3 ubiquitin ligase for DNA-PKcs, promotes apoptosis during DNA damage

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P304

1352-1361

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scholarly article

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10.1038/NM.4415

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11

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23

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Jill S Barnholtz-Sloan

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2017-10-09T00:00:00Z

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1092142130

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29035367

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5679732

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