Accumulation mode particles and LPS exposure induce TLR-4 dependent and independent inflammatory responses in the lung.

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Baicalin Inhibits Haemophilus Parasuis-Induced High-Mobility Group Box 1 Release during Inflammation.In vitro and in vivo defensive effect of probiotic LAB against Pseudomonas aeruginosa using Caenorhabditis elegans model

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Accumulation mode particles and LPS exposure induce TLR-4 dependent and independent inflammatory responses in the lung.

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Accumulation mode particles an ...... mmatory responses in the lung.

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Angela M Fonceca

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Darryl A Knight

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Debra J Turner

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Elizabeth M Bozanich

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Erika N Sutanto

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Paul S McNamara

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P2860

Defective LPS Signaling in C3H/HeJ and C57BL/10ScCr Mice: Mutations in Tlr4 Gene

Toxicity of fibers and particles report of the workshop held in Munich, Germany, October 26-27, 2000

Understanding biophysicochemical interactions at the nano-bio interface

Ambient particle inhalation and the cardiovascular system: potential mechanisms

Derivation of occupational exposure levels (OELs) of low-toxicity isometric biopersistent particles: How can the kinetic lung overload paradigm be used for improved inhalation toxicity study design and OEL-derivation?

Effect of dispersants of multi-walled carbon nanotubes on cellular uptake and biological responses

Outdoor air pollution and asthma

The effect of particle size, location and season on the toxicity of urban and rural particulate matter

Health effects of fine particulate air pollution: lines that connect

Toll-like receptor 4 stimulation before or after Streptococcus pneumoniae induced sepsis improves survival and is dependent on T-cells.

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Accumulation mode particles and LPS exposure induce TLR-4 dependent and independent inflammatory responses in the lung.

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P2860

P2860

Accumulation mode particles and LPS exposure induce TLR-4 dependent and independent inflammatory responses in the lung.

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