CpG DNA redirects class-switching towards "Th1-like" Ig isotype production via TLR9 and MyD88.
about
Toll-like receptors and B-cell receptors synergize to induce immunoglobulin class-switch DNA recombination: relevance to microbial antibody responsesA combined nucleocapsid vaccine induces vigorous SARS-CD8+ T-cell immune responses.Insufficiently defined genetic background confounds phenotypes in transgenic studies as exemplified by malaria infection in Tlr9 knockout miceNew vaccines against influenza virus.Incorporation of membrane-anchored flagellin into influenza virus-like particles enhances the breadth of immune responses.Molecular characterization of in vivo adjuvant activity in ferrets vaccinated against influenza virusIntradermal vaccination with influenza virus-like particles by using microneedles induces protection superior to that with intramuscular immunization.Neural injury following stroke: are Toll-like receptors the link between the immune system and the CNS?Interferon-α accelerates murine systemic lupus erythematosus in a T cell-dependent manner.Toll-like receptor ligands induce expression of the costimulatory molecule CD155 on antigen-presenting cells.Translational mini-review series on Toll-like receptors: Toll-like receptor ligands as novel pharmaceuticals for allergic disorders.Innate signaling networks in mucosal IgA class switchingViral double-stranded RNA triggers Ig class switching by activating upper respiratory mucosa B cells through an innate TLR3 pathway involving BAFF.Innate control of B cell responses.In vivo activation of toll-like receptor-9 induces an age-dependent abortive lytic cycle reactivation of murine gammaherpesvirus-68.TGFβ activated kinase 1 (TAK1) at the crossroad of B cell receptor and Toll-like receptor 9 signaling pathways in human B cells.CpG oligodeoxynucleotide treatment enhances innate resistance and acquired immunity to African trypanosomes.Expansion of polyreactive B cells cross-reactive to HLA and self in the blood of a patient with kidney graft rejectionKinetic Analysis of CpG-Induced Mouse B Cell Growth and Ig ProductionToll-like receptor function in primary B cell defects.A Rapid-Response Humoral Vaccine Platform Exploiting Pre-Existing Non-Cognate Populations of Anti-Vaccine or Anti-Viral CD4+ T Helper Cells to Confirm B Cell Activation.TLR9/MyD88 signaling is required for class switching to pathogenic IgG2a and 2b autoantibodies in SLE.Waddlia chondrophila induces systemic infection, organ pathology, and elicits Th1-associated humoral immunity in a murine model of genital infection.Plasmacytoid dendritic cells and the regulation of immunoglobulin heavy chain class switching.Agonists of Toll-like receptors 3, 4, 7, and 9 are candidates for use as adjuvants in an outer membrane vaccine against Neisseria meningitidis serogroup B.TLR9 and STING agonists synergistically induce innate and adaptive type-II IFN.Toll-like receptors: key activators of leucocytes and regulator of haematopoiesis.Adjuvants are Key Factors for the Development of Future Vaccines: Lessons from the Finlay Adjuvant Platform.B and CD4+ T-cell expression of TLR2 is critical for optimal induction of a T-cell-dependent humoral immune response to intact Streptococcus pneumoniaeTLR9 adjuvants enhance immunogenicity and protective efficacy of the SE36/AHG malaria vaccine in nonhuman primate models.Toll-like receptors--sentries in the B-cell response.Regulation of B-cell responses by Toll-like receptors.Micro and nanoparticle-based delivery systems for vaccine immunotherapy: an immunological and materials perspective.Toll-like receptors and B cells: functions and mechanisms.Toll-like receptors in the pathogenesis of human B cell malignancies.The role of toll-like receptors in B-cell development and immunopathogenesis of common variable immunodeficiency.Characterization of T-Dependent and T-Independent B Cell Responses to a Virus-like Particle.Impact of formulation and particle size on stability and immunogenicity of oil-in-water emulsion adjuvants.Reduced severity of peanut-induced anaphylaxis in TLR9-deficient mice is associated with selective defects in humoral immunity.Strategies for selective priming of memory B cells.
P2860
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P2860
CpG DNA redirects class-switching towards "Th1-like" Ig isotype production via TLR9 and MyD88.
description
2004 nî lūn-bûn
@nan
2004年の論文
@ja
2004年学术文章
@wuu
2004年学术文章
@zh-cn
2004年学术文章
@zh-hans
2004年学术文章
@zh-my
2004年学术文章
@zh-sg
2004年學術文章
@yue
2004年學術文章
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2004年學術文章
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name
CpG DNA redirects class-switch ...... production via TLR9 and MyD88.
@en
CpG DNA redirects class-switch ...... production via TLR9 and MyD88.
@nl
type
label
CpG DNA redirects class-switch ...... production via TLR9 and MyD88.
@en
CpG DNA redirects class-switch ...... production via TLR9 and MyD88.
@nl
prefLabel
CpG DNA redirects class-switch ...... production via TLR9 and MyD88.
@en
CpG DNA redirects class-switch ...... production via TLR9 and MyD88.
@nl
P2093
P356
P1476
CpG DNA redirects class-switch ...... production via TLR9 and MyD88.
@en
P2093
Andrea J Gerth
Stanford L Peng
P304
P356
10.1002/EJI.200324736
P407
P577
2004-05-01T00:00:00Z