COX2 induction: a mechanism of endocrine breast cancer resistance? - Wikidata - awgldk - TriplyDB

COX2 induction: a mechanism of endocrine breast cancer resistance?

COX2 induction: a mechanism of endocrine breast cancer resistance?

http://www.wikidata.org/entity/Q47722410

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COX2 induction: a mechanism of endocrine breast cancer resistance?

http://www.wikidata.org/entity/Q47722410

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2017 nî lūn-bûn

@nan

2017年の論文

@ja

2017年学术文章

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2017年学术文章

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2017年学术文章

@zh-cn

2017年学术文章

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2017年学术文章

@zh-my

2017年学术文章

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2017年學術文章

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2017年學術文章

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name

COX2 induction: a mechanism of endocrine breast cancer resistance?

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COX2 induction: a mechanism of endocrine breast cancer resistance?

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type

label

COX2 induction: a mechanism of endocrine breast cancer resistance?

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COX2 induction: a mechanism of endocrine breast cancer resistance?

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prefLabel

COX2 induction: a mechanism of endocrine breast cancer resistance?

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COX2 induction: a mechanism of endocrine breast cancer resistance?

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S10549-017-4284-7

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Breast Cancer Research and Treatment

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COX2 induction: a mechanism of endocrine breast cancer resistance?

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Brandi L Clark

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Landry K Kamdem

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Michael A Murphy

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P2860

Global cancer statistics

Obesity and breast cancer: role of inflammation and aromatase

Minireview: Inflammation: an instigator of more aggressive estrogen receptor (ER) positive breast cancers

Nonsteroidal anti-inflammatory drugs as anticancer agents: mechanistic, pharmacologic, and clinical issues

Survival and safety of exemestane versus tamoxifen after 2-3 years' tamoxifen treatment (Intergroup Exemestane Study): a randomised controlled trial

Elevated serum estradiol and testosterone concentrations are associated with a high risk for breast cancer. Study of Osteoporotic Fractures Research Group.

Estrogen and the risk of breast cancer.

Cyclooxygenase-2: a target for the prevention and treatment of breast cancer.

Impact of COX2 genotype, ER status and body constitution on risk of early events in different treatment groups of breast cancer patients.

Global cancer statistics in the year 2000.

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s10549-017-4284-7

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383-389

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scholarly article

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10.1007/S10549-017-4284-7

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2

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165

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2017-06-21T00:00:00Z

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28639029

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