Epidermal EGFR controls cutaneous host defense and prevents inflammation.
about
Mechanisms underlying skin disorders induced by EGFR inhibitorsRNase 7 in Cutaneous DefenseCutaneous adverse reactions specific to epidermal growth factor receptor inhibitorsAcneiform Rash Induced by EGFR Inhibitors: Review of the Literature and New InsightsEpidermal RAF prevents allergic skin diseaseEpithelial inflammation resulting from an inherited loss-of-function mutation in EGFRChanges in sebum levels and the development of acneiform rash in patients with non-small cell lung cancer after treatment with EGFR inhibitorsTemporal and gefitinib-sensitive regulation of cardiac cytokine expression via chronic β-adrenergic receptor stimulation.Infection of keratinocytes with Trichophytum rubrum induces epidermal growth factor-dependent RNase 7 and human beta-defensin-3 expression.Dysbiosis and Staphylococcus aureus Colonization Drives Inflammation in Atopic Dermatitis.iRhoms 1 and 2 are essential upstream regulators of ADAM17-dependent EGFR signalingThe Inflammasome and the Epidermal Growth Factor Receptor (EGFR) Are Involved in the Staphylococcus aureus-Mediated Induction of IL-1alpha and IL-1beta in Human Keratinocytes.Nasal vestibulitis due to targeted therapies in cancer patients.Antibiotic prophylaxis for skin toxicity induced by antiepidermal growth factor receptor agents: a systematic review and meta-analysis.Generation and characterization of a target-selectively activated antibody against epidermal growth factor receptor with enhanced anti-tumor potency.Multitasking basal cells: combining stem cell and innate immune duties.The EGF receptor ligand amphiregulin controls cell division via FoxM1Emerging functions of amphiregulin in orchestrating immunity, inflammation, and tissue repair.Preclinical studies of a Pro-antibody-drug conjugate designed to selectively target EGFR-overexpressing tumors with improved therapeutic efficacy.Epidermal growth factor receptor inhibitors trigger a type I interferon response in human skin.Immunological visibility: posttranscriptional regulation of human NKG2D ligands by the EGF receptor pathway.Epidermal growth factor receptor signalling in keratinocyte biology: implications for skin toxicity of tyrosine kinase inhibitors.Biology of the eyelash hair follicle: an enigma in plain sight.Efficacy of recombinant bovine epidermal growth factor in the treatment of experimental subclinical Staphylococcus aureus mastitis in a ewe model.Atypical skin reaction in a patient treated with gefitinib for advanced lung cancer: A case report and review of the literature.EGFR inhibitor-induced skin reactions: differentiating acneiform rash from superimposed bacterial infections.Cell autonomous or systemic EGFR blockade alters the immune-environment in squamous cell carcinomas.Identifying and Creating the Next Generation of Community-Based Cancer Prevention Studies: Summary of a National Cancer Institute Think Tank.EGFR inhibitors erlotinib and lapatinib ameliorate epidermal blistering in pemphigus vulgaris in a non-linear, V-shaped relationship.ADAM17/EGFR axis promotes transglutaminase-dependent skin barrier formation through phosholipase C γ1 and protein kinase C pathways.Mechanisms of skin aging induced by EGFR inhibitors.Preventing or treating anti-EGFR related skin rash with antibiotics?RNase 7 participates in cutaneous innate control of Corynebacterium amycolatum.Genetic ablation of epidermal EGFR reveals the dynamic origin of adverse effects of anti-EGFR therapy.E-cadherin integrates mechanotransduction and EGFR signaling to control junctional tissue polarization and tight junction positioning.Targeted Therapies: Immunologic Effects and Potential Applications Outside of Cancer.Functional Genomics Approach Identifies Novel Signaling Regulators of TGFα Ectodomain Shedding.Epidermal Growth Factor Receptor Signaling Disruption by Endocrine and Metabolic Disrupting Chemicals.Skinflammation and drug toxicity--a delicate balance.Purpuric Drug Eruptions Caused by Epidermal Growth Factor Receptor Inhibitors for Non-Small Cell Lung Cancer: A Clinicopathologic Study of 32 Cases.
P2860
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P2860
Epidermal EGFR controls cutaneous host defense and prevents inflammation.
description
2013 nî lūn-bûn
@nan
2013年の論文
@ja
2013年学术文章
@wuu
2013年学术文章
@zh
2013年学术文章
@zh-cn
2013年学术文章
@zh-hans
2013年学术文章
@zh-my
2013年学术文章
@zh-sg
2013年學術文章
@yue
2013年學術文章
@zh-hant
name
Epidermal EGFR controls cutaneous host defense and prevents inflammation.
@en
Epidermal EGFR controls cutaneous host defense and prevents inflammation.
@nl
type
label
Epidermal EGFR controls cutaneous host defense and prevents inflammation.
@en
Epidermal EGFR controls cutaneous host defense and prevents inflammation.
@nl
prefLabel
Epidermal EGFR controls cutaneous host defense and prevents inflammation.
@en
Epidermal EGFR controls cutaneous host defense and prevents inflammation.
@nl
P2093
P2860
P50
P1476
Epidermal EGFR controls cutaneous host defense and prevents inflammation.
@en
P2093
Andreas Kislat
Andreas Wollenberg
Bernhard Homey
Bettina A Buhren
Edwin Boelke
Holger Schrumpf
Jens M Schröder
Jens W Fischer
Jürgen Harder
Katharina Röck
P2860
P304
P356
10.1126/SCITRANSLMED.3005886
P407
P577
2013-08-01T00:00:00Z