Scn1b deletion leads to increased tetrodotoxin-sensitive sodium current, altered intracellular calcium homeostasis and arrhythmias in murine hearts.
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Sudden unexpected death in epilepsy: Identifying risk and preventing mortalityUltrastructure of the intercellular space in adult murine ventricle revealed by quantitative tomographic electron microscopyLate Na(+) current and protracted electrical recovery are critical determinants of the aging myopathyNeuronal Na+ channel blockade suppresses arrhythmogenic diastolic Ca2+ release.A Regional Reduction in Ito and IKACh in the Murine Posterior Left Atrial Myocardium Is Associated with Action Potential Prolongation and Increased Ectopic Activity.Neuronal Na+ Channels Are Integral Components of Pro-arrhythmic Na+/Ca2+ Signaling Nanodomain That Promotes Cardiac Arrhythmias During β-adrenergic Stimulation.Cardiac arrhythmia in a mouse model of sodium channel SCN8A epileptic encephalopathyNanophysiology: Bridging synapse ultrastructure, biology, and physiology using scanning ion conductance microscopy.Revealing the Concealed Nature of Long-QT Type 3 Syndrome.Voltage-Gated Na+ Channels: Not Just for Conduction.Murine Electrophysiological Models of Cardiac Arrhythmogenesis.Neuronal sodium channels: emerging components of the nano-machinery of cardiac calcium cycling.Changes in cardiac Nav1.5 expression, function, and acetylation by pan-histone deacetylase inhibitors.Sodium and calcium regulation in cardiac myocytes: from molecules to heart failure and arrhythmia.Modulation of Abnormal Sodium Channel Currents in Heart and Brain: Hope for SUDEP Prevention and Seizure Reduction.Voltage-Gated Sodium Channel β Subunits and Their Related Diseases.Voltage-gated sodium channel β subunits: The power outside the pore in brain development and disease.Azithromycin Causes a Novel Proarrhythmic Syndrome.Voltage-Gated Sodium Channel β1/β1B Subunits Regulate Cardiac Physiology and Pathophysiology.Channelopathy as a SUDEP Biomarker in Dravet Syndrome Patient-Derived Cardiac MyocytesCardiac Arrhythmias as Manifestations of Nanopathies: An Emerging View
P2860
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P2860
Scn1b deletion leads to increased tetrodotoxin-sensitive sodium current, altered intracellular calcium homeostasis and arrhythmias in murine hearts.
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name
Scn1b deletion leads to increa ...... arrhythmias in murine hearts.
@en
Scn1b deletion leads to increa ...... arrhythmias in murine hearts.
@nl
type
label
Scn1b deletion leads to increa ...... arrhythmias in murine hearts.
@en
Scn1b deletion leads to increa ...... arrhythmias in murine hearts.
@nl
prefLabel
Scn1b deletion leads to increa ...... arrhythmias in murine hearts.
@en
Scn1b deletion leads to increa ...... arrhythmias in murine hearts.
@nl
P2093
P2860
P50
P1476
Scn1b deletion leads to increa ...... arrhythmias in murine hearts.
@en
P2093
Akshay Shekhar
Chunling Chen
Glenn I Fishman
Heather O'Malley
Mingliang Zhang
Monique Foster
Xianming Lin
P2860
P304
P356
10.1113/JPHYSIOL.2014.277699
P407
P577
2014-09-17T00:00:00Z