Hemolytic Uremic Syndrome in Pregnancy and Postpartum.

Item

http://www.wikidata.org/entity/Q47736433

description

2017 nî lūn-bûn

@nan

2017年の論文

@ja

2017年学术文章

@wuu

2017年学术文章

@zh

2017年学术文章

@zh-cn

2017年学术文章

@zh-hans

2017年学术文章

@zh-my

2017年学术文章

@zh-sg

2017年學術文章

@yue

2017年學術文章

@zh-hant

name

Hemolytic Uremic Syndrome in Pregnancy and Postpartum.

@en

Hemolytic Uremic Syndrome in Pregnancy and Postpartum.

@nl

type

Item

label

Hemolytic Uremic Syndrome in Pregnancy and Postpartum.

@en

Hemolytic Uremic Syndrome in Pregnancy and Postpartum.

@nl

prefLabel

Hemolytic Uremic Syndrome in Pregnancy and Postpartum.

@en

Hemolytic Uremic Syndrome in Pregnancy and Postpartum.

@nl

P1476

Hemolytic Uremic Syndrome in Pregnancy and Postpartum.

@en

P2093

Alexandra Bruel

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Caterina Mele

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Chantal Loirat

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David Kavanagh

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Edwin K S Wong

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Elena Bresin

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Fadi Fakhouri

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François Provôt

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Marina Noris

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Vicky Brocklebank

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P2860

Structural basis for engagement by complement factor H of C3b on a self surface

Relative role of genetic complement abnormalities in sporadic and familial aHUS and their impact on clinical phenotype

Association of factor H autoantibodies with deletions of CFHR1, CFHR3, CFHR4, and with mutations in CFH, CFI, CD46, and C3 in patients with atypical hemolytic uremic syndrome

Complement regulatory proteins at the feto-maternal interface during human placental development: distribution of CD59 by comparison with membrane cofactor protein (CD46) and decay accelerating factor (CD55)

Mutations in components of complement influence the outcome of Factor I-associated atypical hemolytic uremic syndrome

Genetic disorders in complement (regulating) genes in patients with atypical haemolytic uraemic syndrome (aHUS)

Mutations in complement regulatory proteins predispose to preeclampsia: a genetic analysis of the PROMISSE cohort

The binding of factor H to a complex of physiological polyanions and C3b on cells is impaired in atypical hemolytic uremic syndrome.

Structural and functional characterization of factor H mutations associated with atypical hemolytic uremic syndrome

Mutations in factor H reduce binding affinity to C3b and heparin and surface attachment to endothelial cells in hemolytic uremic syndrome.

P304

1237-1247

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P31

scholarly article

P356

10.2215/CJN.00280117

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P433

http://www.w3.org/2001/XMLSchema#string

P478

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P50

Giuseppe Remuzzi

P577

2017-06-08T00:00:00Z

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P698

28596415

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P921

postpartum period

P932

5544502

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