Non-nuclear estrogen receptor alpha activation in endothelium reduces cardiac ischemia-reperfusion injury in mice.

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Ginsenoside Rb1 for Myocardial Ischemia/Reperfusion Injury: Preclinical Evidence and Possible Mechanisms.Notch1 Mediates Preconditioning Protection Induced by GPER in Normotensive and Hypertensive Female Rat Hearts.

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Non-nuclear estrogen receptor alpha activation in endothelium reduces cardiac ischemia-reperfusion injury in mice.

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2017 nî lūn-bûn

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2017年の論文

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2017年学术文章

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Non-nuclear estrogen receptor ...... ia-reperfusion injury in mice.

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Non-nuclear estrogen receptor ...... ia-reperfusion injury in mice.

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label

Non-nuclear estrogen receptor ...... ia-reperfusion injury in mice.

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Non-nuclear estrogen receptor ...... ia-reperfusion injury in mice.

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Non-nuclear estrogen receptor ...... ia-reperfusion injury in mice.

@en

Non-nuclear estrogen receptor ...... ia-reperfusion injury in mice.

@nl

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Non-nuclear estrogen receptor ...... mia-reperfusion injury in mice

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Angel Aponte

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Elizabeth Murphy

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John A Katzenellenbogen

http://www.w3.org/2001/XMLSchema#string

Junhui Sun

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Ken L Chambliss

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Philip W Shaul

http://www.w3.org/2001/XMLSchema#string

Sara Menazza

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Sung Hoon Kim

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Swathi Appachi

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A transmembrane intracellular estrogen receptor mediates rapid cell signaling

Cardiovascular disease outcomes during 6.8 years of hormone therapy: Heart and Estrogen/progestin Replacement Study follow-up (HERS II)

Design of pathway preferential estrogens that provide beneficial metabolic and vascular effects without stimulating reproductive tissues

Activation of a novel estrogen receptor, GPER, is cardioprotective in male and female rats

Estrogen-induced activation of Erk-1 and Erk-2 requires the G protein-coupled receptor homolog, GPR30, and occurs via trans-activation of the epidermal growth factor receptor through release of HB-EGF

Fulvestrant for hormone-sensitive metastatic breast cancer.

Cardioprotection by S-nitrosation of a cysteine switch on mitochondrial complex I.

Non-nuclear estrogen receptor alpha signaling promotes cardiovascular protection but not uterine or breast cancer growth in mice.

The AF-1 activation-function of ERalpha may be dispensable to mediate the effect of estradiol on endothelial NO production in mice

Estrogen action via the G protein-coupled receptor, GPR30: stimulation of adenylyl cyclase and cAMP-mediated attenuation of the epidermal growth factor receptor-to-MAPK signaling axis.

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41-51

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scholarly article

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10.1016/J.YJMCC.2017.04.004

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107

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Benita Katzenellenbogen

Sohaib A Khan

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2017-04-27T00:00:00Z

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28457941

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endothelium

reperfusion injury

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5514412

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Non-nuclear estrogen receptor alpha activation in endothelium reduces cardiac ischemia-reperfusion injury in mice.

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Non-nuclear estrogen receptor alpha activation in endothelium reduces cardiac ischemia-reperfusion injury in mice.

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