Hypoxia induces phosphorylation of the cyclic AMP response element-binding protein by a novel signaling mechanism.
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Identification of ATF-2 as a transcriptional regulator for the tyrosine hydroxylase geneHypoxia-activated metabolic pathway stimulates phosphorylation of p300 and CBP in oxygen-sensitive cellsRegulation of gene expression for neurotransmitters during adaptation to hypoxia in oxygen-sensitive neuroendocrine cells.Hypoxia-induced regulation of MAPK phosphatase-1 as identified by subtractive suppression hybridization and cDNA microarray analysis.Functional genomics approach to hypoxia signaling.Cyclic adenosine monophosphate response element-binding protein phosphorylation and neuroprotection by 4-phenyl-1-(4-phenylbutyl) piperidine (PPBP).Catecholamine biosynthesis and physiological regulation in neuroendocrine cells.Responding to hypoxia: lessons from a model cell line.Ecto-5'-nucleotidase (CD73) regulation by hypoxia-inducible factor-1 mediates permeability changes in intestinal epithelia.Insulin-like growth factor-1 inscribes a gene expression profile for angiogenic factors and cancer progression in breast epithelial cells.Chronic hypoxia-induced morphological and neurochemical changes in the carotid body.The α and Δ isoforms of CREB1 are required to maintain normal pulmonary vascular resistance.Phosphorylation-dependent targeting of cAMP response element binding protein to the ubiquitin/proteasome pathway in hypoxiaAnalysis of expression and posttranslational modification of proteins during hypoxia.HIF1 and oxygen sensing in the brain.Short-term hypoxia transiently increases dopamine β-hydroxylase immunoreactivity in glomus cells of the rat carotid body.Prolyl 4-hydroxylase activity-responsive transcription factors: from hydroxylation to gene expression and neuroprotection.Chronic anemic hypoxemia increases plasma glucagon and hepatic PCK1 mRNA in late-gestation fetal sheep.cAMP-response element-binding protein (CREB) and NF-κB transcription factors are activated during prolonged hypoxia and cooperatively regulate the induction of matrix metalloproteinase MMP1.Mitochondrial (dys)function in adipocyte (de)differentiation and systemic metabolic alterations.Tumor necrosis factor-α potentiates long-term potentiation in the rat dentate gyrus after acute hypoxia.Complex molecular regulation of tyrosine hydroxylase.Hypoxia induces epithelial amphiregulin gene expression in a CREB-dependent manner.CREB-mediated Bcl-2 protein expression after ischemic preconditioning.Human activated macrophages and hypoxia: a comprehensive review of the literature.Respiratory gases and the regulation of transcription.Characterization of a CREB gain-of-function mutant with constitutive transcriptional activity in vivo.Visfatin is present in bovine mammary epithelial cells, lactating mammary gland and milk, and its expression is regulated by cAMP pathway.The von Hippel Lindau/hypoxia-inducible factor (HIF) pathway regulates the transcription of the HIF-proline hydroxylase genes in response to low oxygen.O2-sensitive K+ channels: role of the Kv1.2 -subunit in mediating the hypoxic response.Age-related toxicity of amyloid-beta associated with increased pERK and pCREB in primary hippocampal neurons: reversal by blueberry extract.Basic fibroblast growth factor-induced activation of novel CREB kinase during the differentiation of immortalized hippocampal cells.Hypoxia induces the activation of the phosphatidylinositol 3-kinase/Akt cell survival pathway in PC12 cells: protective role in apoptosis.Oxalate selectively activates p38 mitogen-activated protein kinase and c-Jun N-terminal kinase signal transduction pathways in renal epithelial cells.NMDA receptor-dependent CREB activation in survival of cerebellar granule cells during in vivo and in vitro development.Altered Redox Balance in the Development of Chronic Hypoxia-induced Pulmonary Hypertension.Thyroid Hormone and the Brain: Mechanisms of Action in Development and Role in Protection and Promotion of Recovery after Brain Injury.Neuroprotection by NMDA preconditioning against glutamate cytotoxicity is mediated through activation of ERK 1/2, inactivation of JNK, and by prevention of glutamate-induced CREB inactivation.
P2860
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P2860
Hypoxia induces phosphorylation of the cyclic AMP response element-binding protein by a novel signaling mechanism.
description
1998 nî lūn-bûn
@nan
1998年の論文
@ja
1998年学术文章
@wuu
1998年学术文章
@zh
1998年学术文章
@zh-cn
1998年学术文章
@zh-hans
1998年学术文章
@zh-my
1998年学术文章
@zh-sg
1998年學術文章
@yue
1998年學術文章
@zh-hant
name
Hypoxia induces phosphorylatio ...... y a novel signaling mechanism.
@en
Hypoxia induces phosphorylatio ...... y a novel signaling mechanism.
@nl
type
label
Hypoxia induces phosphorylatio ...... y a novel signaling mechanism.
@en
Hypoxia induces phosphorylatio ...... y a novel signaling mechanism.
@nl
prefLabel
Hypoxia induces phosphorylatio ...... y a novel signaling mechanism.
@en
Hypoxia induces phosphorylatio ...... y a novel signaling mechanism.
@nl
P2860
P921
P356
P1476
Hypoxia induces phosphorylatio ...... y a novel signaling mechanism.
@en
P2093
D Beitner-Johnson
D E Millhorn
P2860
P304
19834-19839
P356
10.1074/JBC.273.31.19834
P407
P577
1998-07-01T00:00:00Z