Spastic long-lasting reflexes in the awake rat after sacral spinal cord injury.
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Recovery of neuronal and network excitability after spinal cord injury and implications for spasticityRecovery of motoneuron and locomotor function after spinal cord injury depends on constitutive activity in 5-HT2C receptors.Transcriptional regulation of gene expression clusters in motor neurons following spinal cord injuryFlexor reflex decreases during sympathetic stimulation in chronic human spinal cord injury.Management of spasticity after spinal cord injury: current techniques and future directions.Vibration-induced extra torque during electrically-evoked contractions of the human calf musclesTreatment of rat spinal cord injury with the neurotrophic factor albumin-oleic acid: translational application for paralysis, spasticity and pain.Characteristics and organization of discharge properties in rat hindlimb motoneurons.Motor neuron firing dysfunction in spastic patients with primary lateral sclerosisPersistent inward currents in spinal motoneurons: important for normal function but potentially harmful after spinal cord injury and in amyotrophic lateral sclerosis.Locomotion after spinal cord injury depends on constitutive activity in serotonin receptorsWind-up of stretch reflexes as a measure of spasticity in chronic spinalized rats: The effects of passive exercise and modafinil.Adrenergic receptors modulate motoneuron excitability, sensory synaptic transmission and muscle spasms after chronic spinal cord injury.Motoneuron excitability and muscle spasms are regulated by 5-HT2B and 5-HT2C receptor activity.Orexin-A modulates glutamatergic NMDA-dependent spinal reflex potentiation via inhibition of NR2B subunit.Down-regulation of KCC2 expression and phosphorylation in motoneurons, and increases the number of in primary afferent projections to motoneurons in mice with post-stroke spasticity.Self-sustained motor activity triggered by interlimb reflexes in chronic spinal cord injury, evidence of functional ascending propriospinal pathways.Polysynaptic excitatory postsynaptic potentials that trigger spasms after spinal cord injury in rats are inhibited by 5-HT1B and 5-HT1F receptorsSensitization of neonatal rat lumbar motoneuron by the inflammatory pain mediator bradykininHip-phase-dependent flexion reflex modulation and expression of spasms in patients with spinal cord injury.Decrease of mRNA Editing after Spinal Cord Injury is Caused by Down-regulation of ADAR2 that is Triggered by Inflammatory ResponseThe transformation of synaptic to system plasticity in motor output from the sacral cord of the adult mouse.Motor axonal regeneration after partial and complete spinal cord transectionRole of endogenous release of norepinephrine in muscle spasms after chronic spinal cord injuryThe swimming test is effective for evaluating spasticity after contusive spinal cord injury.Activation of 5-HT2A receptors upregulates the function of the neuronal K-Cl cotransporter KCC2.Plasticity of connections underlying locomotor recovery after central and/or peripheral lesions in the adult mammalsSpinal cord injury causes plasticity in a subpopulation of lamina I GABAergic interneurons.Intrinsic and synaptic homeostatic plasticity in motoneurons from mice with glycine receptor mutations.Awake behaving electrophysiological correlates of forelimb hyperreflexia, weakness and disrupted muscular synchronization following cervical spinal cord injury in the rat.Activity-dependent plasticity in spinal cord injury.Changes in sensory-evoked synaptic activation of motoneurons after spinal cord injury in man.Active properties of motoneurone dendrites: diffuse descending neuromodulation, focused local inhibition.Intrinsic excitability differs between murine hypoglossal and spinal motoneurons.Adaptive changes in the injured spinal cord and their role in promoting functional recovery.Impaired crossed facilitation of the corticospinal pathway after cervical spinal cord injury.Effects of reversible spinalization on individual spinal neurons.Intramuscular Neurotrophin-3 normalizes low threshold spinal reflexes, reduces spasms and improves mobility after bilateral corticospinal tract injury in rats.Synthesis, transport, and metabolism of serotonin formed from exogenously applied 5-HTP after spinal cord injury in rats.Motor unit.
P2860
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P2860
Spastic long-lasting reflexes in the awake rat after sacral spinal cord injury.
description
2004 nî lūn-bûn
@nan
2004年の論文
@ja
2004年学术文章
@wuu
2004年学术文章
@zh-cn
2004年学术文章
@zh-hans
2004年学术文章
@zh-my
2004年学术文章
@zh-sg
2004年學術文章
@yue
2004年學術文章
@zh
2004年學術文章
@zh-hant
name
Spastic long-lasting reflexes in the awake rat after sacral spinal cord injury.
@en
Spastic long-lasting reflexes in the awake rat after sacral spinal cord injury.
@nl
type
label
Spastic long-lasting reflexes in the awake rat after sacral spinal cord injury.
@en
Spastic long-lasting reflexes in the awake rat after sacral spinal cord injury.
@nl
prefLabel
Spastic long-lasting reflexes in the awake rat after sacral spinal cord injury.
@en
Spastic long-lasting reflexes in the awake rat after sacral spinal cord injury.
@nl
P2093
P2860
P356
P1476
Spastic long-lasting reflexes in the awake rat after sacral spinal cord injury.
@en
P2093
P2860
P304
P356
10.1152/JN.00946.2003
P407
P577
2004-05-01T00:00:00Z