JunD mediates survival signaling by the JNK signal transduction pathway. - Wikidata - awgldk - TriplyDB

JunD mediates survival signaling by the JNK signal transduction pathway.

JunD mediates survival signaling by the JNK signal transduction pathway.

http://www.wikidata.org/entity/Q47811742

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Mammalian sterile 20-like kinase 3 (MST3) mediates oxidative-stress-induced cell death by modulating JNK activation Menin suppresses osteoblast differentiation by antagonizing the AP-1 factor, JunD JAMP, a Jun N-terminal kinase 1 (JNK1)-associated membrane protein, regulates duration of JNK activity.Role for the pleckstrin homology domain-containing protein CKIP-1 in AP-1 regulation and apoptosis A novel vascular disrupting agent plinabulin triggers JNK-mediated apoptosis and inhibits angiogenesis in multiple myeloma cells Transcriptional switch by activating transcription factor 2-derived peptide sensitizes melanoma cells to apoptosis and inhibits their tumorigenicity TNF-stimulated MAP kinase activation mediated by a Rho family GTPase signaling pathway TAK1 is a master regulator of epidermal homeostasis involving skin inflammation and apoptosis Role of FIP200 in cardiac and liver development and its regulation of TNFalpha and TSC-mTOR signaling pathways Pathophysiological significance of c-jun N-terminal kinase in acetaminophen hepatotoxicity Reactive oxygen species in inflammation and tissue injury TAK1, but not TAB1 or TAB2, plays an essential role in multiple signaling pathways in vivo.Jun kinase delays caspase-9 activation by interaction with the apoptosome Enhanced hypoxic preconditioning by isoflurane: signaling gene expression and requirement of intracellular Ca2+ and inositol triphosphate receptors c-Jun N-terminal kinase (JNK)-dependent acute liver injury from acetaminophen or tumor necrosis factor (TNF) requires mitochondrial Sab protein expression in mice c-Jun NH2-terminal kinase is required for lineage-specific differentiation but not stem cell self-renewal.Microarray analysis of Shigella flexneri-infected epithelial cells identifies host factors important for apoptosis inhibition Reduction of Orc6 expression sensitizes human colon cancer cells to 5-fluorouracil and cisplatin Gene expression profiles of bone marrow cells from mice phenotype-selected for maximal or minimal acute inflammations: searching for genes in acute inflammation modifier loci.Sustained c-Jun-NH2-kinase activity promotes epithelial-mesenchymal transition, invasion, and survival of breast cancer cells by regulating extracellular signal-regulated kinase activation.Posttranscriptional downregulation of c-IAP2 by the ubiquitin protein ligase c-IAP1 in vivo Febrile-range hyperthermia augments lipopolysaccharide-induced lung injury by a mechanism of enhanced alveolar epithelial apoptosis CARMA1-mediated NF-kappaB and JNK activation in lymphocytes.JunD/AP-1-mediated gene expression promotes lymphocyte growth dependent on interleukin-7 signal transduction.Role of CHOP in hepatic apoptosis in the murine model of intragastric ethanol feeding Suppression of p53-dependent senescence by the JNK signal transduction pathway JNK regulates FoxO-dependent autophagy in neurons The role of JNK in the development of hepatocellular carcinoma.Disassembly of the JIP1/JNK molecular scaffold by caspase-3-mediated cleavage of JIP1 during apoptosis JNK1 is required for lentivirus entry and gene transfer Tolfenamic Acid Suppresses Inflammatory Stimuli-Mediated Activation of NF-κB Signaling.Hypoxic response contributes to altered gene expression and precapillary pulmonary hypertension in patients with sickle cell disease LEARNING LOCAL DIRECTED ACYCLIC GRAPHS BASED ON MULTIVARIATE TIME SERIES DATA.Requirement of c-Jun NH(2)-terminal kinase for Ras-initiated tumor formation.Adhesion-mediated squamous cell carcinoma survival through ligand-independent activation of epidermal growth factor receptor.Modulation of pancreatic cancer chemoresistance by inhibition of TAK1 Foxo and Fos regulate the decision between cell death and survival in response to UV irradiation.Control of oncogenesis and cancer therapy resistance.Reactive nitrogen species-induced cell death requires Fas-dependent activation of c-Jun N-terminal kinase IL-32α suppresses colorectal cancer development via TNFR1-mediated death signaling.

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P2860

JunD mediates survival signaling by the JNK signal transduction pathway.

http://www.wikidata.org/entity/Q47811742

description

2003 nî lūn-bûn

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2003年の論文

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2003年学术文章

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2003年学术文章

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2003年学术文章

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2003年学术文章

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2003年学术文章

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2003年学术文章

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2003年學術文章

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2003年學術文章

@zh-hant

name

JunD mediates survival signaling by the JNK signal transduction pathway.

@en

JunD mediates survival signaling by the JNK signal transduction pathway.

@nl

type

label

JunD mediates survival signaling by the JNK signal transduction pathway.

@en

JunD mediates survival signaling by the JNK signal transduction pathway.

@nl

prefLabel

JunD mediates survival signaling by the JNK signal transduction pathway.

@en

JunD mediates survival signaling by the JNK signal transduction pathway.

@nl

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JunD mediates survival signaling by the JNK signal transduction pathway.

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Jennifer A Lamb

http://www.w3.org/2001/XMLSchema#string

Juan-Jose Ventura

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Patricia Hess

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Richard A Flavell

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Roger J Davis

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6

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11

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12820962

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