Decreased enteric fatty acid amide hydrolase activity is associated with colonic inertia in slow transit constipation.
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Palmitoylethanolamide normalizes intestinal motility in a model of post-inflammatory accelerated transit: involvement of CB₁ receptors and TRPV1 channels.The importance of defecography in the assessment of the etiology of chronic constipation: An analysis of 630 patients.Toward modulation of the endocannabinoid system for treatment of gastrointestinal disease: FAAHster but not "higher".Endocannabinoids in the gastrointestinal tract.The gastrointestinal tract - a central organ of cannabinoid signaling in health and disease.Cannabinoids and GI Disorders: Endogenous and Exogenous.Targeting fatty acid amide hydrolase and transient receptor potential vanilloid-1 simultaneously to modulate colonic motility and visceral sensation in the mouse: A pharmacological intervention with N-arachidonoyl-serotonin (AA-5-HT).Cannabinoids and gastrointestinal motility: Pharmacology, clinical effects, and potential therapeutics in humans
P2860
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P2860
Decreased enteric fatty acid amide hydrolase activity is associated with colonic inertia in slow transit constipation.
description
2014 nî lūn-bûn
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2014年の論文
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name
Decreased enteric fatty acid a ...... in slow transit constipation.
@en
Decreased enteric fatty acid a ...... in slow transit constipation.
@nl
type
label
Decreased enteric fatty acid a ...... in slow transit constipation.
@en
Decreased enteric fatty acid a ...... in slow transit constipation.
@nl
prefLabel
Decreased enteric fatty acid a ...... in slow transit constipation.
@en
Decreased enteric fatty acid a ...... in slow transit constipation.
@nl
P2093
P2860
P356
P1476
Decreased enteric fatty acid a ...... in slow transit constipation.
@en
P2093
Kai-Lei Jiang
Peng-Jun Su
Shu-Cheng Zhang
Wei-Lin Wang
Zheng-Wei Yuan
P2860
P304
P356
10.1111/JGH.12346
P577
2014-02-01T00:00:00Z