Neuronal lysosomal dysfunction releases exosomes harboring APP C-terminal fragments and unique lipid signatures.

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Potential Effects of MSC-Derived Exosomes in Neuroplasticity in Alzheimer's Disease Glucocerebrosidase deficiency promotes protein aggregation through dysregulation of extracellular vesicles Role of Extracellular Vesicles in Amyotrophic Lateral Sclerosis

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Neuronal lysosomal dysfunction releases exosomes harboring APP C-terminal fragments and unique lipid signatures.

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http://www.wikidata.org/entity/Q47853126

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2018 nî lūn-bûn

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2018年の論文

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2018年学术文章

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name

Neuronal lysosomal dysfunction ...... s and unique lipid signatures.

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Neuronal lysosomal dysfunction ...... s and unique lipid signatures.

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type

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label

Neuronal lysosomal dysfunction ...... s and unique lipid signatures.

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Neuronal lysosomal dysfunction ...... s and unique lipid signatures.

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Neuronal lysosomal dysfunction ...... s and unique lipid signatures.

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Neuronal lysosomal dysfunction ...... s and unique lipid signatures.

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P1476

Neuronal lysosomal dysfunction ...... s and unique lipid signatures.

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P2093

André M Miranda

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Gilbert Di Paolo

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Jessi Neufeld

http://www.w3.org/2001/XMLSchema#string

Robin B Chan

http://www.w3.org/2001/XMLSchema#string

Sabrina Simoes

http://www.w3.org/2001/XMLSchema#string

Sanjid Shahriar

http://www.w3.org/2001/XMLSchema#string

Scott A Small

http://www.w3.org/2001/XMLSchema#string

Yimeng Xu

http://www.w3.org/2001/XMLSchema#string

Zofia M Lasiecka

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P2860

Proteomic and biochemical analyses of human B cell-derived exosomes. Potential implications for their function and multivesicular body formation

Phosphatidylinositol 3,5-bisphosphate: low abundance, high significance

Early etiology of Alzheimer's disease: tipping the balance toward autophagy or endosomal dysfunction?

Depletion of microglia and inhibition of exosome synthesis halt tau propagation.

Proteomic comparison defines novel markers to characterize heterogeneous populations of extracellular vesicle subtypes

Alpha-synuclein induces lysosomal rupture and cathepsin dependent reactive oxygen species following endocytosis

Selective VPS34 inhibitor blocks autophagy and uncovers a role for NCOA4 in ferritin degradation and iron homeostasis in vivo

A highly potent and selective Vps34 inhibitor alters vesicle trafficking and autophagy

Impact of lysosome status on extracellular vesicle content and release

p62/SQSTM1 functions as a signaling hub and an autophagy adaptor

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s41467-017-02533-w

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scholarly article

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10.1038/S41467-017-02533-W

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English

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P478

http://www.w3.org/2001/XMLSchema#string

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Tiago Gil Oliveira

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2018-01-18T00:00:00Z

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29348617

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5773483

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Neuronal lysosomal dysfunction releases exosomes harboring APP C-terminal fragments and unique lipid signatures.

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P2860

P2860

Neuronal lysosomal dysfunction releases exosomes harboring APP C-terminal fragments and unique lipid signatures.

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