Modulation of Navitoclax Sensitivity by Dihydroartemisinin-Mediated MCL-1 Repression in BCR-ABL+ B-Lineage Acute Lymphoblastic Leukemia.

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Antitumor Research on Artemisinin and Its Bioactive Derivatives.

P2860

Q52597265-5D4AA6F7-7F58-4F28-A39A-23E64AFEE5C5

P2860

Modulation of Navitoclax Sensitivity by Dihydroartemisinin-Mediated MCL-1 Repression in BCR-ABL+ B-Lineage Acute Lymphoblastic Leukemia.

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http://www.wikidata.org/entity/Q47858676

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2017 nî lūn-bûn

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2017年の論文

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2017年学术文章

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2017年学术文章

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2017年学术文章

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2017年学术文章

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2017年学术文章

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2017年學術文章

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2017年學術文章

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Modulation of Navitoclax Sensi ...... Acute Lymphoblastic Leukemia.

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Modulation of Navitoclax Sensi ...... Acute Lymphoblastic Leukemia.

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type

Item

label

Modulation of Navitoclax Sensi ...... Acute Lymphoblastic Leukemia.

@en

Modulation of Navitoclax Sensi ...... Acute Lymphoblastic Leukemia.

@nl

prefLabel

Modulation of Navitoclax Sensi ...... Acute Lymphoblastic Leukemia.

@en

Modulation of Navitoclax Sensi ...... Acute Lymphoblastic Leukemia.

@nl

P1476

Modulation of Navitoclax Sensi ...... Acute Lymphoblastic Leukemia.

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P2093

Anisha Kothari

http://www.w3.org/2001/XMLSchema#string

Charles G Mullighan

http://www.w3.org/2001/XMLSchema#string

David Finkelstein

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Ewa Kaminska

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Haiyan Xu

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John C Panetta

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Joseph T Opferman

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Meghan E Turnis

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Michelle L Churchman

http://www.w3.org/2001/XMLSchema#string

Xue Yang

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P2860

The redox antimalarial dihydroartemisinin targets human metastatic melanoma cells but not primary melanocytes with induction of NOXA-dependent apoptosis

Glycogen synthase kinase-3 regulates mitochondrial outer membrane permeabilization and apoptosis by destabilization of MCL-1

Mechanisms of apoptosis sensitivity and resistance to the BH3 mimetic ABT-737 in acute myeloid leukemia

Chemotherapeutic agents circumvent emergence of dasatinib-resistant BCR-ABL kinase mutations in a precise mouse model of Philadelphia chromosome-positive acute lymphoblastic leukemia

A CHOP-regulated microRNA controls rhodopsin expression

Structural insights into the degradation of Mcl-1 induced by BH3 domains

Structure of the BH3 domains from the p53-inducible BH3-only proteins Noxa and Puma in complex with Mcl-1

ABT-199, a potent and selective BCL-2 inhibitor, achieves antitumor activity while sparing platelets

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Gefitinib-induced killing of NSCLC cell lines expressing mutant EGFR requires BIM and can be enhanced by BH3 mimetics

P304

7558-7568

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P31

scholarly article

P356

10.1158/1078-0432.CCR-17-1231

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P407

English

P433

http://www.w3.org/2001/XMLSchema#string

P478

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P50

Amit Budhraja

P577

2017-10-03T00:00:00Z

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P698

28974549

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P932

5786379

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Modulation of Navitoclax Sensitivity by Dihydroartemisinin-Mediated MCL-1 Repression in BCR-ABL+ B-Lineage Acute Lymphoblastic Leukemia.

about

P2860

P2860

Modulation of Navitoclax Sensitivity by Dihydroartemisinin-Mediated MCL-1 Repression in BCR-ABL+ B-Lineage Acute Lymphoblastic Leukemia.

description

name

type

label

prefLabel

P1433

P1476

P2093

P2860

P304

P31

P356

P407

P433

P478

P50

P577

P698

P932

P356

P1433

P1476

P2093

P2860

P304

P31

P356

P407

P433

P478

P50

P577

P698

P932