Inflammasome polymorphisms confer susceptibility to sporadic malignant melanoma.
about
NOD-Like Receptors: Master Regulators of Inflammation and CancerThe inflammasome: an emerging therapeutic oncotarget for cancer preventionData on inflammasome gene polymorphisms of patients with sporadic malignant melanoma in a Brazilian cohort.Identification of rare genetic variation of NLRP1 gene in familial multiple sclerosis.Autolytic proteolysis within the function to find domain (FIIND) is required for NLRP1 inflammasome activity.Genetic Polymorphisms in Inflammasome-Dependent Innate Immunity among Pediatric Patients with Severe Renal Parenchymal Infections.Interactome-wide analysis identifies end-binding protein 1 as a crucial component for the speck-like particle formation of activated absence in melanoma 2 (AIM2) inflammasomes.Inflammasomes in Myeloid Cells: Warriors Within.Inflammasomes in Inflammation-Induced Cancer.Mitophagy or how to control the Jekyll and Hyde embedded in mitochondrial metabolism: implications for melanoma progression and drug resistance.The genetics of innate immunity sensors and human disease.Do inflammatory pathways drive melanomagenesis?NLR-regulated pathways in cancer: opportunities and obstacles for therapeutic interventions.P2X7 receptor and NLRP3 inflammasome activation in head and neck cancer.Inflammasomes and dermatology.Inflammasomes and Cancer.Genetic polymorphisms of IL-18 rs1946518 and IL-1β rs16944 are associated with prognosis and survival of acute myeloid leukemia.Role of inflammasome genetics in susceptibility to HPV infection and cervical cancer development.Ultraviolet Radiation-Induced Downregulation of SERCA2 Mediates Activation of NLRP3 Inflammasome in Basal Cell Carcinoma.NLRP1 Overexpression Is Correlated with the Tumorigenesis and Proliferation of Human Breast Tumor.NLRP3 inflammasome activation plays a carcinogenic role through effector cytokine IL-18 in lymphoma.MiR-22 sustains NLRP3 expression and attenuates H. pylori-induced gastric carcinogenesis.The Emerging Role of Inflammasomes as Central Mediators in Inflammatory Bladder Pathology.Genetic variation and alterations of genes involved in NFκB/TNFAIP3- and NLRP3-inflammasome signaling affect susceptibility and outcome of colorectal cancer.Association of NLRP1 and NLRP3 Polymorphisms with Psoriasis Vulgaris Risk in the Chinese Han Population.
P2860
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P2860
Inflammasome polymorphisms confer susceptibility to sporadic malignant melanoma.
description
2012 nî lūn-bûn
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2012年の論文
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2012年学术文章
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2012年学术文章
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2012年学术文章
@zh-cn
2012年学术文章
@zh-hans
2012年学术文章
@zh-my
2012年学术文章
@zh-sg
2012年學術文章
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2012年學術文章
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name
Inflammasome polymorphisms confer susceptibility to sporadic malignant melanoma.
@en
Inflammasome polymorphisms confer susceptibility to sporadic malignant melanoma.
@nl
type
label
Inflammasome polymorphisms confer susceptibility to sporadic malignant melanoma.
@en
Inflammasome polymorphisms confer susceptibility to sporadic malignant melanoma.
@nl
prefLabel
Inflammasome polymorphisms confer susceptibility to sporadic malignant melanoma.
@en
Inflammasome polymorphisms confer susceptibility to sporadic malignant melanoma.
@nl
P2093
P2860
P1476
Inflammasome polymorphisms confer susceptibility to sporadic malignant melanoma.
@en
P2093
Cecilia Bivik
Charlotta Enerbäck
Deepti Verma
Ensieh Farahani
Inger Rosdahl
Ingrid Synnerstad
Mats Fredrikson
Peter Söderkvist
P2860
P304
P356
10.1111/J.1755-148X.2012.01008.X
P577
2012-06-01T00:00:00Z